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The effect of non-antihypertensive doses of angiotensin converting enzyme inhibitor on myocardial necrosis and hypertrophy in young rats with renovascular hypertension
(1999-05-06)
In renovascular hypertensive rats, low doses of angiotensin converting enzyme (ACE) inhibitors have been found to prevent myocardial hypertrophy independent of blood pressure level. This finding would suggest humoral rather ...
MEF2C Silencing Attenuates Load-Induced Left Ventricular Hypertrophy by Modulating mTOR/S6K Pathway in Mice
(Public Library ScienceSan FranciscoEUA, 2009)
Effects of lisinopril on experimental ischemia in rats. Influence of infarct size
(Sociedade Brasileira de Cardiologia (SBC), 1999-10-01)
OBJECTIVE - Angiotensin-converting enzyme inhibitors (ACEIs) have gained importance in preventing or attenuating the process of ventricular remodeling after myocardial infarction. The significance of infarct size in regard ...
Effects of nandrolone and resistance training on the blood pressure, cardiac electrophysiology, and expression of atrial beta-adrenergic receptors
(Pergamon-elsevier Science LtdOxfordInglaterra, 2013)
Cardiac hypertrophy reduction in SHR by specific silencing of myocardial Na/H exchanger
(American Physiological Society, 2015-03)
We examined the effect of specific and local silencing of sodium/hydrogen exchanger isoform 1 (NHE1) with a small hairpin RNA delivered by lentivirus (L-shNHE1) in the cardiac left ventricle (LV) wall of spontaneously ...
Ace gene dosage influences the development of renovascular hypertension
(WILEY-BLACKWELL PUBLISHING, INC, 2010)
P>1. Clinical and experimental evidence highlights the importance of the renin-angiotensin system in renovascular hypertension. Furthermore, genetic factors affecting angiotensin-converting enzyme (ACE) could influence the ...
Ca2+/ Calmodulin dependent protein kinase II and androgen signaling pathways modulate MEF2 activity in testosterone induced cardiac myocyte hypertrophy
(Frontiers media SA, 2017)
Testosterone is known to induce cardiac hypertrophy through androgen receptor (AR)-dependent and -independent pathways, but the molecular underpinnings of the androgen action remain poorly understood. Previous work has ...