Article
Side Fenestrations Provide an "anchor" for a Stable Binding of A1899 to the Pore of TASK-1 Potassium Channels
Fecha
2017Registro en:
14, 7, 2197-2208
15438384
Autor
Ramírez D.
Arévalo B.
Martínez G.
Rinné S.
Sepúlveda F.V.
Decher N.
González W.
Institución
Materias
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Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
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Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
School of Medicine; Universidade de São Paulo (USP); University of Edinburgh; Edificio 17 Piano Terra; University of Pittsburgh; Universidade Estadual Paulista (Unesp); SHM B147 (2006-07-12)Type II Bartter's syndrome is a hereditary hypokalemic renal salt-wasting disorder caused by mutations in the ROMK channel (Kir1.1; Kcnj1), mediating potassium recycling in the thick ascending limb of Henle's loop (TAL) ... -
Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
School of Medicine; Universidade de São Paulo (USP); University of Edinburgh; Edificio 17 Piano Terra; University of Pittsburgh; Universidade Estadual Paulista (Unesp); SHM B147 (2006-07-12)Type II Bartter's syndrome is a hereditary hypokalemic renal salt-wasting disorder caused by mutations in the ROMK channel (Kir1.1; Kcnj1), mediating potassium recycling in the thick ascending limb of Henle's loop (TAL) ...