Review
Possible role of mitochondrial permeability transition pore in the pathogenesis of Huntington disease
Fecha
2017Registro en:
483, 4, 1078-1083
0006291X
Autor
Quintanilla R.A.
Tapia C.
Pérez M.J.
Institución
Materias
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Rosiglitazone treatment prevents mitochondrial dysfunction in mutant huntingtinexpressing cells: possible role of peroxisome proliferator-activated receptor-gamma (PPARgamma) in the pathogenesis of Huntington disease.
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