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Mostrando ítems 1431-1440 de 1440
FOXG1 Regulates PRKAR2B Transcriptionally and Posttranscriptionally via miR200 in the Adult Hippocampus
(Humana Press, 2019-07)
Rett syndrome is a complex neurodevelopmental disorder that is mainly caused by mutations in MECP2. However, mutations in FOXG1 cause a less frequent form of atypical Rett syndrome, called FOXG1 syndrome. FOXG1 is a key ...
Tomato I2 immune receptor can be engineered to confer partial resistance to the oomycete Phytophthora infestans in addition to the fungus Fusarium oxysporum
(American Phytopathological Society, 2015-09)
Plants and animals rely on immune receptors, known as nucleotide-binding domain and leucine-rich repeat containing proteins (NB-LRR or NLR), to defend against invading pathogens and activate immune responses. How NLR ...
Histamine H2 receptor trafficking: role of arrestin, dynamin and clathrin in histamine H2 receptor internalization
(American Society for Pharmacology and Experimental Therapeutics, 2008)
Encephalization and diversification of the cranial base in platyrrhine primates
(Academic Press Ltd - Elsevier Science Ltd, 2015)
Gonadotropin- releasing hormone agonist affects rat ovarian follicle development by interfering with FSH and growth factors on the prevention of apoptosis
(Wiley-liss, Div John Wiley & Sons Inc, 2001)
Adsorption of R-OH molecules on TiO2 surfaces at the solid-liquid interface
(American Chemical Society, 2011)
Hexachlorobenzene induces cell proliferation, and aryl hydrocarbon receptor expression (AhR) in rat liver preneoplastic foci, and in the human hepatoma cell line HepG2. AhR is a mediator of ERK1/2 signaling, and cell cycle regulation in HCB-treated HepG2 cells
(Elsevier Ireland, 2015-10)
Hexachlorobenzene (HCB) is a widespread environmental pollutant, and a liver tumor promoter in rodents. Depending on the particular cell lines studied, exposure to these compounds may lead to cell proliferation, terminal ...
Critical role of mTOR, PPARγ and PPARδ signaling in regulating early pregnancy decidual function, embryo viability and feto-placental growth
(Oxford University Press, 2018-06)
STUDY QUESTION: What are the consequences of inhibiting mTOR, the mechanistic target of rapamycin (mTOR), and the peroxisome proliferator activated receptor gamma (PPARγ) and PPARδ pathways in the early post-implantation ...