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Effects of MPP+ on the molecular pathways involved in cell cycle control in B65 neuroblastoma cells
(2010)
The toxicity caused by cell exposure to 1-methyl-4-phenylpyridinium ion (MPP+) is a useful model in the study of Parkinson's disease (PD). However, the exact molecular mechanisms triggered by MPP+ in cell death are currently ...
Lack of Jun-N-terminal kinase 3 (JNK3) does not protect against neurodegeneration induced by 3-nitropropionic acid
(2012)
Aims: 3-Nitropropionic acid (3-NP) is a toxin that replicates most of the clinical and pathophysiological symptoms of Huntington's disease, inducing neurodegeneration in the striatum due to the inhibition of mitochondrial ...
Effects of MPP+ on the molecular pathways involved in cell cycle control in B65 neuroblastoma cells
(2010)
The toxicity caused by cell exposure to 1-methyl-4-phenylpyridinium ion (MPP+) is a useful model in the study of Parkinson's disease (PD). However, the exact molecular mechanisms triggered by MPP+ in cell death are currently ...
Lack of Jun-N-terminal kinase 3 (JNK3) does not protect against neurodegeneration induced by 3-nitropropionic acid
(2012)
Aims: 3-Nitropropionic acid (3-NP) is a toxin that replicates most of the clinical and pathophysiological symptoms of Huntington's disease, inducing neurodegeneration in the striatum due to the inhibition of mitochondrial ...