Rhabdomyolysis and severe hypercalcemia. Report of one case

dc.creatorHuidobro E, Juan Pablo
dc.creatorSantander, Jaime
dc.creatorVicentini, Daniel
dc.creatorJara, Aquiles
dc.date.accessioned2024-01-10T12:42:10Z
dc.date.accessioned2024-05-02T16:50:27Z
dc.date.available2024-01-10T12:42:10Z
dc.date.available2024-05-02T16:50:27Z
dc.date.created2024-01-10T12:42:10Z
dc.date.issued2019
dc.identifier0717-6163
dc.identifier0034-9887
dc.identifierMEDLINE:30848776
dc.identifierhttps://repositorio.uc.cl/handle/11534/77487
dc.identifierWOS:000460163000019
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/9267206
dc.description.abstractRhabdomyolysis (RD) is the process that leads to cell destruction of striated muscle. Causes include inherited metabolic defects or acquired disorders. RD is frequently associated with acute kidney injury (AKI) and disorders of calcium metabolism. We report a 33 year old man that after amphetamine consumption and an uninterrupted 3,000 km driving presented vomiting, muscle pain and dark urine. He had elevated creatinkinase levels, severe hypocalcemia and an acute renal failure. He was treated with hemodialysis and calcitriol. He was transferred to our hospital and on admission a serum calcium of 18 mg/dl was detected. He continued on hemodialysis, recovering renal function and with normalization of creatinkinase levels and serum calcium level.
dc.languagees
dc.publisherSOC MEDICA SANTIAGO
dc.rightsregistro bibliográfico
dc.subjectAcute Kidney Injury
dc.subjectHypercalcemia
dc.subjectRhabdomyolysis
dc.subjectACUTE-RENAL-FAILURE
dc.subjectDELAYED HYPERCALCEMIA
dc.titleRhabdomyolysis and severe hypercalcemia. Report of one case
dc.typeartículo


Este ítem pertenece a la siguiente institución