Article
Reversion of the Ballantyne syndrome despite fetal hydrops persistence
Registro en:
LOBATO, Gustavo; NAKAMURA-PEREIRA, Marcos. Reversion of the Ballantyne Syndrome despite Fetal Hydrops Persistence. Fetal Diagnosis and Therapy, v. 24, p. 474-477, 2008.
1015-3837
10.1159/000178532
Autor
Lobato, Gustavo
Nakamura-Pereira, Marcos
Resumen
Introdução: A síndrome de Ballantyne (ou síndrome do espelho) é uma hipertensão proteica gestacional associada à hidropisia fetal. Este relatório descreve um caso em que a reversão da síndrome de Ballantyne ocorreu apesar da persistência da hidropisia fetal. Relato de Caso: Uma mulher de 24 anos apresentou hidropisia fetoplacentária com 28 2/7 semanas de gestação. Aloimunização grave de Rh (D) e anemia hemolítica fetal (hematócrito fetal 15,4%) foram confirmadas por cordocentese e foi realizada transfusão intra-uterina. Ela também revelou hipertensão (160/100 mm Hg), edema e proteinúria (845 mg / dia). Após quatro transfusões intra-uterinas, a pressão arterial foi normalizada; a proteinúria urinária não foi significativa e o edema desapareceu completamente. A hidropisia fetal persistiu até o parto em 32 semanas gestacionais, mas uma redução parcial da hidropisia placentária foi observada. Discussão: A redução total ou parcial do edema placentário pode ser responsável pela reversão da síndrome de Ballantyne, apesar da persistência da hidropisia fetal. Introduction: The Ballantyne syndrome (or mirror syndrome) is a gestational proteinuric hypertension associated with fetal hydrops. This report describes a case in which Ballantyne syndrome reversion occurred despite fetal hydrops persistence. Case Report: A 24-year-old woman showed fetoplacental hydrops at 28 2/7 gestational weeks. Severe Rh(D) alloimmunization and fetal hemolytic anemia (fetal hematocrit 15.4%) were confirmed by cordocentesis, and an intrauterine transfusion was performed. She also revealed hypertension (160/100 mm Hg), edema and proteinuria (845 mg/day). After four intrauterine transfusions, blood pressure was normalized; urinary proteinuria was not significant, and the edema vanished completely. Fetal hydrops persisted until delivery at 32 gestational weeks, but a partial reduction of placental hydrops was noted. Discussion: Total or partial reduction of the placental edema may be responsible for the reversal of the Ballantyne syndrome despite the fetal hydrops persistence. 2200-01-01
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