Tnf-a increases production of reactive oxygen species through cdk5 activation in nociceptive neurons

Frontiers in Physiology

dc.creatorSandoval, Rodrigo
dc.creatorLazcano, Pablo
dc.creatorFerrari, Franco
dc.creatorPinto-Pardo, Nicolás
dc.creatorGonzález-Billault, Christian Enrique
dc.creatorUtreras, Elias
dc.date2018-11-29T15:35:54Z
dc.date2022-07-07T15:33:45Z
dc.date2014
dc.date2018-11-29T15:35:54Z
dc.date2022-07-07T15:33:45Z
dc.date2018
dc.date.accessioned2023-08-21T23:30:24Z
dc.date.available2023-08-21T23:30:24Z
dc.identifier1140325
dc.identifier1140325
dc.identifierhttps://hdl.handle.net/10533/228003
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/8295734
dc.descriptionThe participation of reactive oxygen species (ROS) generated by NOX1 and NOX2/NADPH oxidase has been documented during inflammatory pain. However, the molecular mechanism involved in their activation is not fully understood. We reported earlier a key rol
dc.descriptionRegular
dc.descriptionFONDECYT
dc.descriptionFONDECYT
dc.languageeng
dc.relationhandle/10533/111556
dc.relationhandle/10533/111541
dc.relationhandle/10533/108045
dc.relationhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC5808211/
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.rightsinfo:eu-repo/semantics/openAccess
dc.titleTnf-a increases production of reactive oxygen species through cdk5 activation in nociceptive neurons
dc.titleFrontiers in Physiology
dc.typeArticulo
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion


Este ítem pertenece a la siguiente institución