PLOS ONE;
PLOS ONE

dc.creatorValladares, Denisse
dc.creatorAlmarza, Gonzalo
dc.creatorContreras, Ariel
dc.creatorPavez, Mario
dc.creatorBuvinic, Sonja
dc.creatorJaimovich, Enrique
dc.creatorCasas, Mariana
dc.date2016-12-05T14:55:59Z
dc.date2022-07-06T17:22:14Z
dc.date2016-12-05T14:55:59Z
dc.date2022-07-06T17:22:14Z
dc.date2013
dc.date.accessioned2023-08-21T21:00:20Z
dc.date.available2023-08-21T21:00:20Z
dc.identifier11100454
dc.identifier11100454
dc.identifier1932-6203
dc.identifierhttps://hdl.handle.net/10533/138468
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/8281687
dc.description7
dc.descriptionFONDECYT
dc.descriptionmcasas@med.uchile.cl
dc.description11
dc.descriptione75340
dc.descriptionFONDECYT
dc.description8
dc.languageeng
dc.publisherPUBLIC LIBRAY OF SCIENCE
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.relationinfo:eu-repo/grantAgreement/Fondecyt/11100454
dc.relationinfo:eu-repo/semantics/dataset/hdl.handle.net/10533/93477
dc.relationhttps://doi.org/10.1371/journal.pone.0075340
dc.rightsinfo:eu-repo/semantics/openAccess
dc.titleElectrical stimuli are anti-apoptotic in skeletal muscle via extracellular atp. Alteration of this signal in mdx mice is a likely cause of dystrophy
dc.titlePLOS ONE
dc.titlePLOS ONE
dc.typeArticulo
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.coverageSAN FRANCISCO


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