dc.creator | Díaz-Peña R. | |
dc.creator | Valdés E. | |
dc.creator | Cofré C. | |
dc.creator | Castro-Santos P. | |
dc.date.accessioned | 2020-09-02T22:16:51Z | |
dc.date.accessioned | 2022-11-08T20:22:14Z | |
dc.date.available | 2020-09-02T22:16:51Z | |
dc.date.available | 2022-11-08T20:22:14Z | |
dc.date.created | 2020-09-02T22:16:51Z | |
dc.date.issued | 2015 | |
dc.identifier | 107, 9, 560-566 | |
dc.identifier | 11300108 | |
dc.identifier | https://hdl.handle.net/20.500.12728/4360 | |
dc.identifier.uri | https://repositorioslatinoamericanos.uchile.cl/handle/2250/5144337 | |
dc.language | en | |
dc.language | es | |
dc.publisher | ARAN Ediciones S.A. | |
dc.subject | Autophagy | |
dc.subject | Crohn’s disease | |
dc.subject | Genome-wide association studies | |
dc.subject | Inflammatory bowel disease | |
dc.subject | Th17 | |
dc.subject | Ulcerative colitis | |
dc.subject | caspase recruitment domain protein 15 | |
dc.subject | granulocyte macrophage colony stimulating factor | |
dc.subject | interleukin 10 | |
dc.subject | interleukin 21 | |
dc.subject | interleukin 23 | |
dc.subject | toll like receptor | |
dc.subject | transforming growth factor | |
dc.subject | tumor necrosis factor alpha | |
dc.subject | autophagy | |
dc.subject | bacterial flora | |
dc.subject | cell differentiation | |
dc.subject | diet | |
dc.subject | disease course | |
dc.subject | environmental factor | |
dc.subject | genetic association | |
dc.subject | genetic polymorphism | |
dc.subject | genetic regulation | |
dc.subject | genetic risk | |
dc.subject | genetic susceptibility | |
dc.subject | health care | |
dc.subject | heritability | |
dc.subject | human | |
dc.subject | immune response | |
dc.subject | inflammatory bowel disease | |
dc.subject | meta analysis (topic) | |
dc.subject | prevalence | |
dc.subject | protein expression | |
dc.subject | Review | |
dc.subject | risk assessment | |
dc.subject | signal transduction | |
dc.subject | single nucleotide polymorphism | |
dc.subject | Th17 cell | |
dc.title | Th17 response and autophagy - Main pathways implicated in the development of inflammatory bowel disease by genome-wide association studies: New factors involved in inflammatory bowel disease susceptibility [Respuesta Th17 y autofagia: Principales vías implicadas en enfermedad inflamatoria intestinal por los estudios de asociación de genoma completo: Nuevos factores implicados en la susceptibilidad a enfermedad inflamatoria intestinal] | |
dc.type | Review | |