Prevalence of Listeria monocytogenes in raw milk in Guadalajara, Mexico

Abstract

Congenital palmar polyonychia (CPP) is a rare and usually sporadic birth defect. We report on a Mexican girl with CPP of both 5th fingers and her sister with ectrodactyly with ulnar ray deficits and agenesis of the ulna. In previous reports, CPP has been seen in ectrodactyly with involvement of the ulna and ulnar digital rays and with postaxial polydactyly. Such findings observed in our patients can be considered a form of CPP. Autosomal dominant inheritance of CPP is more likely based in previous informative families with vertical transmission and instances of male-to-male transmission. The present and two previous families with affected sibs only may represent parental gonadal mosaicism, whereas de novo mutation or incomplete evaluation of relatives could be an explanation for sporadic cases. Variable expression of the CPP phenotype may be a new autosomal dominant entity, i.e., a CPP-postaxial limb defect syndrome. " 2003 Wiley-Liss, Inc.",,,,,,,,,"http://hdl.handle.net/20.500.12104/43897","http://www.scopus.com/inward/record.url?eid=2-s2.0-1442308547&partnerID=40&md5=f881078ba82acbf9b8fb110853fd16ec

http://ovidsp.ovid.com/ovidweb.cgi?T=JS&CSC=Y&NEWS=N&PAGE=fulltext&D=med5&AN=14981725",,,,,,"2",,"American Journal of Medical Genetics",,"205

209",,"125 A",,"Scopus

MEDLINE

WOS",,,,"Index Medicus;Adolescent;Female;Fingers/ab [Abnormalities];Foot Deformities, Congenital/ge [Genetics];Foot Deformities, Congenital/pa [Pathology];Hand Deformities, Congenital/ge [Genetics];Hand Deformities, Congenital/pa [Pathology];Humans;Infant;Infectious Disease Transmission, Vertical;Limb Deformities, Congenital/ge [Genetics];Limb Deformities, Congenital/pa [Pathology];Mexico;Mosaicism;Phenotype;Polydactyly/ge [Genetics];Polydactyly/pa [Pathology];Siblings;Syndrome",,"Ectopic nails; Ectrodactyly; Palmar nails; Postaxial polydactyly; Ulnar agenesis",,,,,,"Probable New Syndrome in a Mexican Family with Congenital Palmar Polyonychi and Postaxial Limb Defects",,"Article" "45715","123456789/35008","Junyent, F., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain, Unitat de Bioquimica, Facultat de Medicina i Ciències de la Salut, CIBERNED, Universitat Rovira i Virgili, C./ St. Llorenç 21 43201 Reus, Tarragona, Spain; Alvira, D., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain; Yeste-Velasco, M., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain; de la Torre, A.V., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain; Beas-Zarate, C., Lab. de Desarrollo y Regeneración Neural, Instituto de Neurobiología, Departamento de Biología Celular y Molecular, CUCBA, Universidad de Guadalajara, Guadalajara, Jalisco, Mexico; Sureda, F.X., Unitat de Farmacología, CIBERNED, Facultat de Medicina i Ciències de la Salut, Univ. Rovira i Virgili, C./ St. Llorenç 21 43201 Reus, Tarragona, Spain; Folch, J., Unitat de Bioquimica, Facultat de Medicina i Ciències de la Salut, CIBERNED, Universitat Rovira i Virgili, C./ St. Lloreç 21 43201 Reus, Tarragona, Spain; Pallàs, M., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain; Camins, A., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain; Verdaguer, E., Unitat de Farmacologia i Farmacognòsia, Institut de Biomedicina (IBUB), CIBERNED, Facultat de Farmàcia, Universitat de Barcelona, Nucli Universitari de Pedralbes, 08028 Barcelona, Spain

Beas-Zárate, Carlos., Universidad de Guadalajara. Centro Universitario de Ciencias Biológicas y Agropecuarias",,"Junyent, F.

Alvira, D.

Yeste-Velasco, M.

de la Torre, A.V.

Beas-Zárate, Carlos

Sureda, F.X.

Folch, J.

Pallas, M.

Camins, A.

Verdaguer, E.",,"2010",,"In the present study the role of JAK/STAT and Akt in apoptosis was evaluated in cerebellar granule cells after treatment with the mitochondrial toxin MPP +. Firstly, we evaluated the role of the prosurvival Akt pathway in MPP +-induced apoptosis and found that MPP + rapidly reduced the phosphorylation of Akt at Ser473. Since PTEN is an upstream regulator of Akt, its inhibition with bpV(pic) (1-30?M) should activate Akt, however, it did not attenuate CGC cell death mediated by MPP + but protected CGC from apoptosis mediated by S/K deprivation. We also demonstrated that after the treatment with the complex I inhibitor, the expression levels of STAT1 increased and the levels of STAT3 decreased at the time points tested (0.5-8h). Meanwhile, pharmacological inhibition of the JAK/STAT pathway with AG490 (10-40?M) was neuroprotective, probably due to its antioxidant properties, the Jak2-inhibitor-II potentiated MPP + neurotoxicity. Collectively, our data indicate that the treatment of CGC with the neurotoxin MPP + decreased two prosurvival pathways: STAT3 and Akt. Meanwhile Akt activation, using a PTEN inhibitor, did not play a prominent role in neuroprotection; loss of STAT3 could be a signal pathway involved in neuroprotection against the Parkinsonian neurotoxin MPP +. © 2010 Elsevier Ltd.",,,,,,"10.1016/j.neuint.2010.08.015",,,,"http://www.scopus.com/inward/record.url?eid=2-s2.0-77958107729&partnerID=40&md5=e46fc3dd456e5ab3b3fca5e0bd2b2ac4

http://hdl.handle.net/20.500.12104/43936",,,,,,"7",,"Neurochemistry International",,"774

782",,"57",,"Scopus

WOS",,,,,,"Akt; Apoptosis; MPP +; STAT3, Cerebellar granule cells",,,,"Prosurvival role of JAK/STAT and Akt signaling pathways in MPP +-induced apoptosis in neurons",,,,"Article", "45662","123456789/35008",,"Meza-Espinoza, J.P., Facultad de Medicina, Unidad Académica de Ciencias de la Salud y Tecnología, Universidad Autónoma de Tamaulipas, Tamaulipas, Mexico, División de Genética, CIBO-IMSS, Universidad de Guadalajara, Sierra Mojada No. 800, 44340 Guadalajara, Jalisco, Mexico; Gutiérrez-Angulo, M., División de Genética, CIBO-IMSS, Universidad de Guadalajara, Sierra Mojada No. 800, 44340 Guadalajara, Jalisco, Mexico; Vázquez-Cárdenas, A., División de Genética, CIBO-IMSS, Universidad de Guadalajara, Sierra Mojada No. 800, 44340 Guadalajara, Jalisco, Mexico; Delgado-Lamas, J.L., Departamento de Hematología, Hospital de Especialidades, CMNO-IMSS, Mexico; Esparza-Flores, M.A., Departamento de Hematología, Hospital de Pediatría, CMNO-IMSS, Mexico; González-García, J.R., División de Genética, CIBO-IMSS, Universidad de Guadalajara, Sierra Mojada No. 800, 44340 Guadalajara, Jalisco, Mexico",,"Meza-Espinoza, J.P.

Gutierrez-Angulo, M.

Vazquez-Cardenas, A.

Delgado-Lamas, J.L.

Esparza-Flores, M.A.

Gonzalez-García, J.R.",,"2007",,"RT-PCR studies in 93 patients with chronic myelogenous leukemia from the Mexican West were done in order to know the proportion of b2a2 and b3a2 BCR/ABL1 transcripts. Forty-five patients showed the b3a2 transcript (48%), 37 (40%) displayed the b2a2 and in 11 cases (12%) both transcripts were detected. Statistical analyses showed that these figures are in accordance with two of three similar studies realized in Mexican population. Moreover, significant differences were found among Mexican people and patients from other countries, namely Ecuador, England, Italy, Poland, Japan, and Thailand. Ecuadorian patients showed differences with all the populations analyzed. These variations could be due to a different genetic background.",,,,,,,,,"http://hdl.handle.net/20.500.12104/43883","http://www.scopus.com/inward/record.url?eid=2-s2.0-38049025186&partnerID=40&md5=65d49aa96509a8d92d3dcb844296246b",,,,,,"5",,"Revista de Investigacion Clinica",,"338

341",,"59",,"Scopus

WOS",,,,,,"B2a2 and b3a2 transcripts; Chronic myelogenous leukemia; Mexican patients",,,,,,"Prevalence of the BCR/ABL1 transcripts in Mexican patients with chronic myelogenous leukemia",,"Article" "45720","123456789/35008","Del Angel-Meza, A.R., Laboratorio de Ciencias de los alimentos, Universidad de Guadalajara, Departamento de Reproducción Humana, Crecimiento y Desarrollo Infantil, Centro Universitario de Ciencias de la Salud (CUCS), Guadalajara, Jalisco, Mexico; Dávalos-Marín, A.J., Laboratorio de neuroinmunomodulación, Sierra Mojada # 800, Mexicano del, División de Neurociencias, Colonia Independencia, (IMSS), Instituto, Centro de Investigación Biomédica de Occidente (CIBO), Seguro Social, CP 44340, Guadalajara, Jalisco, Mexico, Departamento de Bioquiamica, Facultad de Medicina, Universidad Autoanoma de Guadalajara (UAG), Guadalajara, Jalisco, Mexico; Ontiveros-Martinez, L.L., Laboratorio de Ciencias de los alimentos, Universidad de Guadalajara, Departamento de Reproducción Humana, Crecimiento y Desarrollo Infantil, Centro Universitario de Ciencias de la Salud (CUCS), Guadalajara, Jalisco, Mexico; Ortiz, G.G., Laboratorio de desarrollo y Envejecimiento, Seguro Social (IMSS), Division de Neurociencias, Centro de Investigacion, Biomedica de Occidente (CIBO), Instituto Mexicano del, Guadalajara, Jalisco, Mexico; Beas-Zarate, C., Laboratorio de Neurobiologia Molecular, Divisioan de Neurociencias, Centro de Investigación Biomédica de Occidente (CIBO), Instituto Mexicano del Seguro Social (IMSS), Guadalajara, Jalisco, Mexico; Chaparro-Huerta, V., Laboratorio de Neurobiologia Molecular, Divisioan de Neurociencias, Centro de Investigación Biomédica de Occidente (CIBO), Instituto Mexicano del Seguro Social (IMSS), Guadalajara, Jalisco, Mexico; Torres-Mendoza, B.M., Laboratorio de Inmunodeficiencias y Retrovirus, Division de Neurociencias, Centro de Investigacion Biomédica de Occidente (CIBO), Instituto Mexicano del Seguro Social (IMSS), Guadalajara, Jalisco, Mexico, Departamento de Clínicas Meadicas, Centro Universitario de Ciencias de la Salud (CUCS), Universidad de Guadalajara, Guadalajara, Jalisco, Mexico; Bitzer-Quintero, O.K., Laboratorio de neuroinmunomodulación, Sierra Mojada # 800, Mexicano del, División de Neurociencias, Colonia Independencia, (IMSS), Instituto, Centro de Investigación Biomédica de Occidente (CIBO), Seguro Social, CP 44340, Guadalajara, Jalisco, Mexico

Beas-Zárate, Carlos., Universidad de Guadalajara. Centro Universitario de Ciencias Biológicas y Agropecuarias",,"Del Angel-Meza, A.R.

Davalos-Marin, A.J.

Ontiveros-Martinez, L.L.

Ortiz, G.G.

Beas-Zárate, Carlos

Chaparro-Huerta, V.

Torres-Mendoza, B.M.

Bitzer-Quintero, O.K.",,"2011",,"Tryptophan (TRP), which plays an important role in immune system regulation, protein synthesis, serotonin (5-HT) and melatonin production, is a potent endogenous free radical scavenger and antioxidant. The aim of this work was to determine the efficacy of TRP in neuro-inflammation induced by systemic administration of lipopolysacharide (LPS, 20. mg/kg) which promotes the synthesis of free radical (LPO: MDA and 4-HDA), and pro-inflammatory cytokine Interferon-? (IFN-?) in different brain regions (cerebral cortex and hippocampus) of rats. Experiments were performed on adult female, pregnant and lactating rats fed with a diet of TRP content (0.5. mg/100. g protein), cerebral cortex and hippocampus were evaluated for lipid peroxidation (LPO) products, nitrites, nitrates and plasmatic concentration of IFN-?. LPO levels in LPS+TRP groups were significantly decreased than that obtained in the LPS group. However, there were no observed differences in plasmatic levels of nitrites and nitrates as well as IFN-?, neither in the cerebral cortex or hippocampus. The TRP has protective effect in the oxidative damage in a model of endotoxic shock in the breading nurslings induced by the systemic administration of LPS, acting as a scavenger of free radicals. So, it can be proposed as an innocuous protector agent in the endotoxic shock process. © 2011 Elsevier Masson SAS.",,,,,,"10.1016/j.biopha.2011.02.008",,,,"http://www.scopus.com/inward/record.url?eid=2-s2.0-79959641835&partnerID=40&md5=70924d509343fef20ad11873424d39af

http://ovidsp.ovid.com/ovidweb.cgi?T=JS&CSC=Y&NEWS=N&PAGE=fulltext&D=medl&AN=21616633

http://hdl.handle.net/20.500.12104/43941",,,,,,"3",,"Biomedicine and Pharmacotherapy",,"215

219",,"65",,"Scopus

MEDLINE

WOS",,,,"Index Medicus;Animals;Antioxidants/pd [Pharmacology];Cerebral Cortex/de [Drug Effects];Cerebral Cortex/me [Metabolism];Drug Interactions;Female;Free Radical Scavengers/me [Metabolism];Free Radical Scavengers/pd [Pharmacology];Hippocampus/de [Drug Effects];Hippocampus/me [Metabolism];Inflammation/bl [Blood];Inflammation/dt [Drug Therapy];Inflammation/me [Metabolism];Interferon-gamma/bl [Blood];Lactation;Lipid Peroxidation/de [Drug Effects];Lipopolysaccharides/pd [Pharmacology];Neuroprotective Agents/pd [Pharmacology];Nitrates/bl [Blood];Nitrites/bl [Blood];Pregnancy;Rats;Rats, Sprague-Dawley;Shock, Septic/bl [Blood];Shock, Septic/dt [Drug Therapy];Shock, Septic/me [Metabolism];Tryptophan/pd [Pharmacology]",,"Central nervous system; Cytokines; Lipopolysaccharide; Melatonin; Tryptophan",,,,"Protective effects of tryptophan on neuro-inflammation in rats after administering lipopolysaccharide",,,,"Article", "45654","123456789/35008",,"Luisjuan-Morales, A., Public Health Laboratory, Regional Center for Research in Public Health, University of Guadalajara, Apartado Postal 2-136, Guadalajara, Jalisco Mexico, 44280, Mexico; Alaniz-De La O, R., Public Health Laboratory, Regional Center for Research in Public Health, University of Guadalajara, Apartado Postal 2-136, Guadalajara, Jalisco Mexico, 44280, Mexico; Vázquez-Sandoval, M.E., Public Health Laboratory, Regional Center for Research in Public Health, University of Guadalajara, Apartado Postal 2-136, Guadalajara, Jalisco Mexico, 44280, Mexico; Rosas-Barbosa, B.T., Public Health Laboratory, Regional Center for Research in Public Health, University of Guadalajara, Apartado Postal 2-136, Guadalajara, Jalisco Mexico, 44280, Mexico",,"Luisjuan-Morales, A.

Alaniz-De La O, R.

Vazquez-Sandoval, M.E.

Rosas-Barbosa, B.T.",,"1995",,"The frequency of L. monocytogenes and other species of Listeria was determined in 100 samples of raw milk obtained from street vendors and retail stores located in the city of Guadalajara, Mexico. Listeria innocua and Listeria welshimeri were isolated from 7 and 2 samples, respectively, whereas L. monocytogenes was not isolated from any samples. Copyright ", International Association of Milk, Food and Environmental Sanitarians.