Secretion of IL-16 through TNFR1 and calpain-caspase signaling contributes to MRSA pneumonia

dc.date.accessioned2020-04-20T19:47:25Z
dc.date.accessioned2022-10-18T23:09:01Z
dc.date.available2020-04-20T19:47:25Z
dc.date.available2022-10-18T23:09:01Z
dc.date.created2020-04-20T19:47:25Z
dc.date.issued2014
dc.identifierhttp://hdl.handle.net/10533/241696
dc.identifier1140010
dc.identifierWOS:000344091700008
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4473030
dc.description.abstractStaphylococcus aureus is a major cause of severe pneumonia. Multiple mechanisms of proinflammatory signaling are activated to recruit immune cells into the airway in response to S. aureus. We found that interleukin-16 (IL-16), a T cell cytokine that bind
dc.languageeng
dc.relationhttps://doi.org/10.1038/mi.2014.24
dc.relation10.1038/mi.2014.24
dc.titleSecretion of IL-16 through TNFR1 and calpain-caspase signaling contributes to MRSA pneumonia
dc.typeArticulo


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