Necroptosis Promotes Staphylococcus aureus Clearance by Inhibiting Excessive Inflammatory Signaling

dc.date.accessioned2020-04-20T19:47:23Z
dc.date.accessioned2022-10-18T23:08:47Z
dc.date.available2020-04-20T19:47:23Z
dc.date.available2022-10-18T23:08:47Z
dc.date.created2020-04-20T19:47:23Z
dc.date.issued2016
dc.identifierhttp://hdl.handle.net/10533/241667
dc.identifier1140010
dc.identifierWOS:000382310100018
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4473001
dc.description.abstractStaphylococcus aureus triggers inflammation through inflammasome activation and recruitment of neutrophils, responses that are critical for pathogen clearance but are associated with substantial tissue damage. We postulated that necroptosis, cell death me
dc.languageeng
dc.relationhttps://doi.org/10.1016/j.celrep.2016.07.039
dc.relation10.1016/j.celrep.2016.07.039
dc.titleNecroptosis Promotes Staphylococcus aureus Clearance by Inhibiting Excessive Inflammatory Signaling
dc.typeArticulo


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