From birth to death: A role for reactive oxygen species in neuronal development

dc.date.accessioned2020-03-11T20:34:33Z
dc.date.accessioned2022-10-18T22:58:12Z
dc.date.available2020-03-11T20:34:33Z
dc.date.available2022-10-18T22:58:12Z
dc.date.created2020-03-11T20:34:33Z
dc.date.issued2017
dc.identifierhttp://hdl.handle.net/10533/240274
dc.identifier15150012
dc.identifierWOS:000435040400007
dc.identifierno scielo
dc.identifiereid=2-s2.0-85029223828
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4471613
dc.description.abstractHistorically, ROS have been considered toxic molecules, especially when their intracellular concentration reaches high values. However, physiological levels of ROS support crucial cellular processes, acting as second messengers able to regulate intrinsic
dc.languageeng
dc.relationhttps://doi.org/10.1016/j.semcdb.2017.09.012
dc.relation10.1016/j.semcdb.2017.09.012
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleFrom birth to death: A role for reactive oxygen species in neuronal development
dc.typeArticulo


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