Prion Function and Pathophysiology in Non-Mammalian Models

dc.date.accessioned2020-03-11T20:34:15Z
dc.date.accessioned2022-10-18T22:57:35Z
dc.date.available2020-03-11T20:34:15Z
dc.date.available2022-10-18T22:57:35Z
dc.date.created2020-03-11T20:34:15Z
dc.date.issued2017
dc.identifierhttp://hdl.handle.net/10533/240199
dc.identifier15150012
dc.identifierWOS:000399790900003
dc.identifierno scielo
dc.identifiereid=2-s2.0-85019562237
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4471538
dc.description.abstractMore than thirty years have passed since the discovery of the prion protein (PrP) and its causative role in transmissible spongiform encephalopathy. Since a combination of both gain- and loss-of-function mechanisms may underlay prion pathogenesis, underst
dc.languageeng
dc.relationhttps://doi.org/10.2174/1566524017666170220100715
dc.relation10.2174/1566524017666170220100715
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titlePrion Function and Pathophysiology in Non-Mammalian Models
dc.typeArticulo


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