Endoplasmic Reticulum-Mitochondria Calcium Communication and the Regulation of Mitochondrial Metabolism in Cancer: A Novel Potential Target

dc.date.accessioned2020-03-11T20:34:14Z
dc.date.accessioned2022-10-18T22:57:33Z
dc.date.available2020-03-11T20:34:14Z
dc.date.available2022-10-18T22:57:33Z
dc.date.created2020-03-11T20:34:14Z
dc.date.issued2017
dc.identifierhttp://hdl.handle.net/10533/240194
dc.identifier15150012
dc.identifierWOS:000410216900001
dc.identifierno scielo
dc.identifiereid=2-s2.0-85029788916
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4471533
dc.description.abstractCancer is characterized by an uncontrolled cell proliferation rate even under low nutrient availability, which is sustained by a metabolic reprograming now recognized as a hallmark of cancer. Warburg was the first to establish the relationship between can
dc.languageeng
dc.relationhttps://doi.org/10.3389/fonc.2017.00199
dc.relation10.3389/fonc.2017.00199
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleEndoplasmic Reticulum-Mitochondria Calcium Communication and the Regulation of Mitochondrial Metabolism in Cancer: A Novel Potential Target
dc.typeArticulo


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