Endoplasmic Reticulum Stress and the Hallmarks of Cancer

dc.date.accessioned2020-03-11T20:34:03Z
dc.date.accessioned2022-10-18T22:57:17Z
dc.date.available2020-03-11T20:34:03Z
dc.date.available2022-10-18T22:57:17Z
dc.date.created2020-03-11T20:34:03Z
dc.date.issued2016
dc.identifierhttp://hdl.handle.net/10533/240158
dc.identifier15150012
dc.identifierWOS:000432223300006
dc.identifierno scielo
dc.identifiereid=2-s2.0-84968918504
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4471497
dc.description.abstractTumor cells are often exposed to intrinsic and external factors that alter protein homeostasis, thus producing endoplasmic reticulum (ER) stress. To cope with this, cells evoke an adaptive mechanism to restore ER proteostasis known as the unfolded protein
dc.languageeng
dc.relationhttps://doi.org/10.1016/j.trecan.2016.03.007
dc.relation10.1016/j.trecan.2016.03.007
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleEndoplasmic Reticulum Stress and the Hallmarks of Cancer
dc.typeArticulo


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