dc.date.accessioned2020-03-11T20:28:20Z
dc.date.accessioned2022-10-18T22:44:50Z
dc.date.available2020-03-11T20:28:20Z
dc.date.available2022-10-18T22:44:50Z
dc.date.created2020-03-11T20:28:20Z
dc.date.issued2014
dc.identifierhttp://hdl.handle.net/10533/238536
dc.identifier15110111
dc.identifierWOS:000334107500113
dc.identifierno scielo
dc.identifiereid=2-s2.0-84899418224
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4469875
dc.description.abstractConsiderable evidence indicates that the NMDA receptor (NMDAR) subunits NR2A and NR2B are critical mediators of synaptic plasticity and dendritogenesis; however, how they differentially regulate these processes is unclear. Here we investigate the roles of
dc.languageeng
dc.relationhttps://doi.org/10.1371/journal.pone.0094037
dc.relation10.1371/journal.pone.0094037
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titlePSD-95 suppresses dendritic arbor development in mature hippocampal neurons by occluding the clustering of NR2B-NMDA receptors
dc.typeArticulo


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