| dc.date.accessioned | 2019-03-27T15:57:23Z | |
| dc.date.accessioned | 2022-10-18T22:09:13Z | |
| dc.date.available | 2019-03-27T15:57:23Z | |
| dc.date.available | 2022-10-18T22:09:13Z | |
| dc.date.created | 2019-03-27T15:57:23Z | |
| dc.date.issued | 2016 | |
| dc.identifier | http://hdl.handle.net/10533/233893 | |
| dc.identifier | 1140108 | |
| dc.identifier.uri | https://repositorioslatinoamericanos.uchile.cl/handle/2250/4465250 | |
| dc.description.abstract | Introduction: Excitotoxicity, i.e. neuronal death by excessive NMDA receptor activation, neuronal Ca2+ overload and nitric oxide (NO) synthesis, is a prominent mechanism leading to cell death (Sandoval et al. 2011, J Neurochem 118:760). NO leads to protei | |
| dc.language | eng | |
| dc.relation | 46° | |
| dc.relation | info:eu-repo/grantAgreement/Fondecyt/1140108 | |
| dc.relation | Annual Meeting of the Society for Neuroscience | |
| dc.rights | info:eu-repo/semantics/openAccess | |
| dc.title | NF-kB S-nitrosylation in excitotoxicity | |
| dc.type | Ponencia | |
