dc.date.accessioned2018-12-07T13:33:39Z
dc.date.accessioned2022-10-18T21:59:34Z
dc.date.available2018-12-07T13:33:39Z
dc.date.available2022-10-18T21:59:34Z
dc.date.created2018-12-07T13:33:39Z
dc.date.issued2016
dc.identifierhttp://hdl.handle.net/10533/232645
dc.identifier1150377
dc.identifierWOS:000370457100031
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4464000
dc.description.abstractThe alpha2-adrenergic receptor agonist Dexmedetomidine (Dex) is a sedative medication used by anesthesiologists. Dex protects the heart against ischemia-reperfusion (IR) and can also act as a preconditioning mimetic. The mechanisms involved in Dex-depend
dc.languageeng
dc.relationhttps://www.ncbi.nlm.nih.gov/pubmed/26607864
dc.relationhandle/10533/111557
dc.relation10.1016/j.phrs.2015.11.004
dc.relationhandle/10533/111541
dc.relationhandle/10533/108045
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleDexmedetomidine Protects The Heart Against Ischemia-Reperfusion Injury By An Endothelial Enos/No Dependent Mechanism
dc.typeArticulo


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