dc.date.accessioned2018-12-07T13:32:41Z
dc.date.accessioned2022-10-18T21:54:15Z
dc.date.available2018-12-07T13:32:41Z
dc.date.available2022-10-18T21:54:15Z
dc.date.created2018-12-07T13:32:41Z
dc.date.issued2016
dc.identifierhttp://hdl.handle.net/10533/231969
dc.identifier1150638
dc.identifierWOS:000371203200002
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4463324
dc.description.abstractThe use of transgenic models for the study of neurodegenerative diseases has made valuable contributions to the field. However, some important limitations, including protein overexpression and general systemic compensation for the missing genes, has caus
dc.languageeng
dc.relationhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC4769573/
dc.relationhandle/10533/111557
dc.relation10.1186/s40659-016-0072-9
dc.relationhandle/10533/111541
dc.relationhandle/10533/108045
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleGenetic variants associated with neurodegenerative alzheimer disease in natural models
dc.typeArticulo


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