Hipokalemia, hipovolemia y repercusión electrocardiográfica secundarias a ingesta prolongada de furosemida: Caso clínico

dc.creatorAravena,César
dc.creatorSalas,Ignacio
dc.creatorTagle,Rodrigo
dc.creatorJara,Aquiles
dc.creatorMiranda,Rodrigo
dc.creatorMcNab,Paul
dc.creatorRodríguez,José A
dc.creatorValdés,Gloria
dc.creatorValdivieso,Andrés
dc.date2007-11-01
dc.date.accessioned2017-03-07T15:58:52Z
dc.date.available2017-03-07T15:58:52Z
dc.identifierhttp://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872007001100013
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/393908
dc.descriptionHypokalemia (serum K+ < 3.5 mEq/1) is a potentially serious adverse effect of diuretic ingestión. We report a 27 year-old woman admitted with muscle weakness, a serum potassium of 2.0 mEq/1, metabolic alkalosis and EKG abnormalities simulating cardiac ischemia, that reverted with potassium chloride administration. She admitted high dose furosemide self-medication for edema. Glomerular filtration rate, tubular sodium reabsortion, potassium secretion, the renin-aldosterone system, total body water distribution and capillary permeability, were studied sequentially until 90 days after her admission. There was hyperactivity of the renin-aldosterone axis, reduction in extracellular and intracellular volumes, normal capillary permeability and high sodium tubular reabsorption, probably explained by a "rebound" salt retention associated with her decreased extracellular volume
dc.formattext/html
dc.languagees
dc.publisherSociedad Médica de Santiago
dc.sourceRevista médica de Chile v.135 n.11 2007
dc.subjectFurosemide
dc.subjectHypokalemia
dc.subjectHypovolemia
dc.titleHipokalemia, hipovolemia y repercusión electrocardiográfica secundarias a ingesta prolongada de furosemida: Caso clínico
dc.typeArtículos de revistas


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