dc.creatorPoblete Durán, Natalia
dc.creatorPrades Pérez, Yara
dc.creatorVera Otarola, Jorge
dc.creatorSoto Rifo, Ricardo
dc.creatorValiente Echeverria, Fernando Andres
dc.date.accessioned2016-12-30T17:34:49Z
dc.date.available2016-12-30T17:34:49Z
dc.date.created2016-12-30T17:34:49Z
dc.date.issued2016
dc.identifierViruses-Basel. Volumen: 8 Número: 7 Número de artículo: 180
dc.identifier10.3390/v8070180
dc.identifierhttps://repositorio.uchile.cl/handle/2250/142228
dc.description.abstractAfter viral infection, host cells respond by mounting an anti-viral stress response in order to create a hostile atmosphere for viral replication, leading to the shut-off of mRNA translation (protein synthesis) and the assembly of RNA granules. Two of these RNA granules have been well characterized in yeast and mammalian cells, stress granules (SGs), which are translationally silent sites of RNA triage and processing bodies (PBs), which are involved in mRNA degradation. This review discusses the role of these RNA granules in the evasion of anti-viral stress responses through virus-induced remodeling of cellular ribonucleoproteins (RNPs).
dc.languageen
dc.publisherMDPI AG
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.rightsAttribution-NonCommercial-NoDerivs 3.0 Chile
dc.sourceViruses-Basel
dc.subjecttranslation control
dc.subjectanti-viral host immune response
dc.subjectP-bodies
dc.subjectstress granules
dc.subjectRNA granules
dc.titleWho Regulates Whom? An Overview of RNA Granules and Viral Infections
dc.typeArtículo de revista


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