Epidermal growth factor stimulates urokinase-type plasminogen activator expression in human gingival fibroblasts. Possible modulation by genistein and curcumin

dc.creatorSmith Schuster, Patricio C.
dc.creatorSantibañez, Juan F.
dc.creatorMorales, Juan P.
dc.creatorMartínez, Jorge
dc.date.accessioned2014-01-10T13:49:47Z
dc.date.available2014-01-10T13:49:47Z
dc.date.created2014-01-10T13:49:47Z
dc.date.issued2004-04-08
dc.identifierJ Periodont Res 2004; 39; 380–387
dc.identifierdoi: 10.1111/j.1600-0765.2004.00753.x
dc.identifierhttps://repositorio.uchile.cl/handle/2250/123507
dc.description.abstractRegulation of the extracellular matrix turnover is a crucial process in wound healing and the progress of periodontal disease. It has been proposed that urokinase-type plasminogen activator (uPA), under the control of growth factors or cytokines, provides the proteolytic potential to the accomplishment of these cellular events. Epidermal growth factor (EGF) is one of the growth factors that has been shown to be active in uPA regulation.
dc.languageen
dc.publisherBlackwell Munksgaard Ltd
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.rightsAttribution-NonCommercial-NoDerivs 3.0 Chile
dc.subjectc-Jun N-terminal kinase; curcumin; epidermal growth factor; genistein; gingival fibroblasts; urokinase
dc.titleEpidermal growth factor stimulates urokinase-type plasminogen activator expression in human gingival fibroblasts. Possible modulation by genistein and curcumin
dc.typeArtículo de revista


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