Chile | Articulo

Crosstalk between the upr and autophagy pathway contributes to handling cellular stress in neurodegenerative disease

dc.date.accessioned2016-12-05T15:23:08Z
dc.date.available2016-12-05T15:23:08Z
dc.date.created2016-12-05T15:23:08Z
dc.date.issued2012
dc.identifier1554-8627
dc.identifierhttp://hdl.handle.net/10533/140706
dc.identifier3100039
dc.identifierWOS:000307193400011
dc.languageeng
dc.publisherLANDES BIOSCIENCE
dc.relationhttps://doi.org/10.4161/auto.20139
dc.relation10.4161/auto.20139
dc.relationinfo:eu-repo/grantAgreement/Fondecyt/3100039
dc.relationinfo:eu-repo/semantics/dataset/hdl.handle.net/10533/93477
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.rightsinfo:eu-repo/semantics/openAccess
dc.titleCrosstalk between the upr and autophagy pathway contributes to handling cellular stress in neurodegenerative disease
dc.typeArticulo


Este ítem pertenece a la siguiente institución