dc.date.accessioned2016-11-29T19:53:08Z
dc.date.available2016-11-29T19:53:08Z
dc.date.created2016-11-29T19:53:08Z
dc.date.issued2011
dc.identifier1071-9164
dc.identifierhttp://hdl.handle.net/10533/129069
dc.identifier1050768
dc.identifierWOS:000298208300007
dc.languageeng
dc.publisherCHURCHILL LIVINGSTONE INC MEDICAL PUBLISHERS
dc.relationhttps://doi.org/10.1016/j.cardfail.2011.08.008
dc.relation10.1016/j.cardfail.2011.08.008
dc.relationinfo:eu-repo/grantAgreement/Fondecyt/1050768
dc.relationinfo:eu-repo/semantics/dataset/hdl.handle.net/10533/93477
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.rightsinfo:eu-repo/semantics/openAccess
dc.titleSystemic oxidative stress and endothelial dysfunction is associated with an attenuated acute vascular response to inhaled prostanoid in pulmonary artery hypertension patients
dc.typeArticulo


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