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Effects of BAY 41-2272, an activator of nitric oxide-independent site of soluble guanylate cyclase, on human NADPH oxidase system from THP-1 cells
(Elsevier Science BvAmsterdamHolanda, 2007)
Contraction-related stimuli regulate GLUT4 traffic in C2C 12-GLUT4myc skeletal muscle cells
(2010)
Muscle contraction stimulates glucose uptake acutely to increase energy supply, but suitable cellular models that faithfully reproduce this complex phenomenon are lacking. To this end, we have developed a cellular model ...
Thyroid hormone activates rat liver adenosine 5-monophosphate-activated protein kinase: Relation to CaMKKβ, TAK1, and LKB1 expression and energy status
(2013)
AMP-activated protein kinase (AMPK) is a sensor of energy status supporting cellular energy homeostasis that may represent the metabolic basis for 3,3′,5-triiodo-L-thyronine (T3) liver preconditioning. Functionally transient ...
Lithium Reversibly Inhibits Schwann Cell Proliferation and Differentiation Without Inducing Myelin Loss
(Humana Press, 2016-12)
This study was undertaken to examine the bioactivity, specificity, and reversibility of lithium?s action on the growth, survival, proliferation, and differentiation of cultured Schwann cells (SCs). In isolated SCs, lithium ...
Caracterizações farmacológicas e bioquímicas de peçonhas de aranhas dos gêneros Lasiodora e Loxosceles
(Universidade Federal de Minas GeraisUFMG, 2012-11-23)
Spider venoms are complex mixtures of substances. Research into spider venoms investigates the mechanisms underlying envenomation, new therapeutic strategies and the huge pharmacological potential of the venom components. ...
Low-level laser therapy (LLLT) acts as cAMP-elevating agent in acute respiratory distress syndrome
(Springer Verlag (Germany), 2017)
Murine myeloid progenitor responses to GM-CSF and eosinophil precursor responses to IL-5 represent distinct targets for downmodulation by prostaglandin E2
(British Pharmacological Society, 2013)
Murine myeloid progenitor responses to GM-CSF and eosinophil precursor responses to IL-5 represent distinct targets for downmodulation by prostaglandin E2
(British Pharmacological Society, 2000)