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Fructose and nonalcoholic fatty liver disease
(Royal Society of Chemistry, 2012)
Nonalcoholic fatty liver disease (NAFLD) is an important emerging public health problem which is expanding worldwide. NAFLD is the liver component of the metabolic syndrome, frequently associated with obesity, type 2 ...
Fructose ingestion impairs expression of genes involved in skeletal muscle’s adaptive response to aerobic exercise
(BioMed Central, 2017)
Abstract
Background
The inverse relationship between exercise capacity and its variation over time and both cardiovascular and all-cause mortality suggests the existence ...
Opposite lipemic response of Wistar rats and C57BL/6 mice to dietary glucose or fructose supplementation
(Associação Brasileira de Divulgação Científica, 2007)
Dietary fructose promotes prostate cancer growth
(2021-06-01)
Clinical localization of primary tumors and sites of metastasis by PET is based on the enhanced cellular uptake of 2-deoxy-2-[18F]-fluoro-D-glucose (FDG). In prostate cancer, however, PET-FDG imaging has shown limited ...
Alpha-lipoic acid and its protective role in fructose induced endocrine-metabolic disturbances
(Royal Society of Chemistry, 2019-10)
In recent decades a worldwide increase has been reported in the consumption of unhealthy high calorie diets associated with marked changes in meal nutrient composition, such as a higher intake of refined carbohydrates, ...
Dietary Fructose Increases the Sensitivity of Proximal Tubules to Angiotensin II in Rats Fed High-Salt Diets
(Multidisciplinary Digital Publishing Institute, 2018-09)
Dietary fructose causes salt-sensitive hypertension. Proximal tubules (PTs) reabsorb 70% of the filtered NaCl. Angiotensin II (Ang II), atrial natriuretic peptide (ANP) and norepinephrine (NE) regulate this process. Although ...
Catechin and quercetin attenuate adipose inflammation in fructose‐fed rats and 3T3‐L1 adipocytes
(Wiley, 2015-04)
Scope
This study evaluated the capacity of dietary catechin (C), quercetin (Q), and the combination of both (CQ), to attenuate adipose inflammation triggered by high fructose (HFr) consumption in rats and by tumor necrosis ...
Dietary (-)-epicatechin affects NF-kappa;B activation and NADPH oxidases in the kidney cortex of high-fructose-fed rats
(Royal Society of Chemistry, 2019-01)
Inflammation involves the activation of redox-sensitive transcription factors, e.g., nuclear factor κB (NF-κB). Administration of (−)-epicatechin to high-fructose-fed rats prevented NF-κB activation and up-regulation of ...
Dietary (-)-epicatechin affects NF-kB activation and NADPH-oxidases in the kidney cortex of high fructose-fed rats
(Royal Society of Chemistry, 2019-02)
Inflammation involves the activation of redox-sensitive transcription factors, e.g. nuclear factor kB (NF-kB). Administration of (-)-epicatechin to high fructose-fed rats prevented NF-kB activation, and up-regulation of ...
Modifications in nitric oxide and superoxide anion metabolism induced by fructose overload in rat heart are prevented by (−)-epicatechin
(Royal Society of Chemistry, 2016-04)
Fructose overload promotes functional and metabolic derangements in humans and in animal experimental models. Evidence suggests that dietary flavonoids have the ability to prevent/attenuate the development of metabolic ...