Hiperglicemia na intoxicação escorpiônica experimental em cão

dc.creatorMaia, Lineu Freire
dc.creatorFerreira, Marcelo Carvalho
dc.creatorSilva, Carlos Gomes da
dc.date2022-07-15T12:04:55Z
dc.date2022-07-15T12:04:55Z
dc.date1959
dc.date.accessioned2023-09-26T22:58:13Z
dc.date.available2023-09-26T22:58:13Z
dc.identifierMAIA, Lineu Freire; FERREIRA, Marcelo Carvalho; SILVA, Carlos Gomes da. Hiperglicemia na intoxicação escorpiônica experimental em cão. Memórias do Instituto Oswaldo Cruz, Rio de Janeiro, v. 57, n. 1, p. 1 - 9, 1959.
dc.identifier0074-0206
dc.identifierhttps://www.arca.fiocruz.br/handle/icict/53827
dc.identifier10.1590/S0074-02761959000100008
dc.identifier1678-8060
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/8885344
dc.descriptionThe production of hyperglycemia during the acute phase of scorpion poisoning produced by T. bahiensis in dogs is confirmed now. The highest degree on average, was reached 10 minutes after the injection of venom. In our hands, the previous bilateral adrenalectomy did not avoid the hyperglycemia. The average of the blood sugar level has been similar to that observed in dogs with adrenal glands, the highest blood sugar level was also registered after 10 minutes. The hyperglycemia obtained in adrenolectomized dogs is, probably, due to the liberation of Sympatin (Nor-adrenalin and adrenalin) as a consequence of the central excitation by the poison on the hepatic nerves and other ganglionar terminations of the Sympathetic Nervous System. Our present researches suggest that the venom has adrenergic action besides the central action.
dc.formatapplication/pdf
dc.languagepor
dc.publisherFundação Oswaldo Cruz. Instituto Oswaldo Cruz.
dc.rightsopen access
dc.subjectHiperglicemia
dc.subjectIntoxicação
dc.subjectEscorpiônica experimental
dc.subjectCão
dc.titleHiperglicemia na intoxicação escorpiônica experimental em cão
dc.typeArticle


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