dc.creatorReis, Roberta Souza dos
dc.creatorHorn, Fabiana
dc.date2012-01-24T01:20:11Z
dc.date2010
dc.identifier1757-4749
dc.identifierhttp://hdl.handle.net/10183/36811
dc.identifier000783194
dc.descriptionA successful infection of the human intestine by enteropathogenic bacteria depends on the ability of bacteria to attach and colonize the intestinal epithelium and, in some cases, to invade the host cell, survive intracellularly and disseminate from cell to cell. To accomplish these processes bacteria have evolved an arsenal of molecules that are mostly secreted by dedicated type III secretion systems, and that interact with the host, subverting normal cellular functions. Here we overview the most important molecular strategies developed by enteropathogenic Escherichia coli, Salmonella enterica, Shigella flexneri, and Yersinia enterocolitica to cause enteric infections. Despite having evolved different effectors, these four microorganisms share common host cellular targets.
dc.formatapplication/pdf
dc.languageeng
dc.relationGut Pathogens. Bethesda: National Center for Biotechnology Information, 2009-. Vol. 2, art. 8 (22 jul. 2010), p. 1-12
dc.rightsOpen Access
dc.subjectEscherichia coli
dc.subjectInfecções bacterianas
dc.subjectSalmonella
dc.subjectShigella flexneri
dc.subjectYersinia enterocolitica
dc.subjectMicrobiologia molecular
dc.titleEnteropathogenic Escherichia coli, Samonella[sic], Shigella and Yersinia : cellular aspects of host-bacteria interactions in enteric diseases
dc.typeArtigo de periódico
dc.typeEstrangeiro


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