APP signaling in Alzheimer's disease

dc.creatorBignante, Elena Anahi
dc.creatorLorenzo, Alfredo Guillermo
dc.date.accessioned2020-02-07T18:08:29Z
dc.date.accessioned2022-10-15T13:51:23Z
dc.date.available2020-02-07T18:08:29Z
dc.date.available2022-10-15T13:51:23Z
dc.date.created2020-02-07T18:08:29Z
dc.date.issued2018-11
dc.identifierBignante, Elena Anahi; Lorenzo, Alfredo Guillermo; APP signaling in Alzheimer's disease; Impact Journals; Aging; 10; 11; 11-2018; 3063-3064
dc.identifier1945-4589
dc.identifierhttp://hdl.handle.net/11336/96883
dc.identifierCONICET Digital
dc.identifierCONICET
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4393475
dc.description.abstractA large body of evidence supports the Amyloid β (Aβ) cascade hypothesis underlying neurodegeneration in Alzheimer’s disease (AD). Although the mechanism by which Aβ induces neuronal dysfunction and death is still matter of debate, in the last two decades several groups have generated compelling evidence supporting a role of Amyloid β Precursor Protein (APP) as a bona fide receptor for Aβ that can trigger neurodegeneration.
dc.languageeng
dc.publisherImpact Journals
dc.relationinfo:eu-repo/semantics/altIdentifier/doi/http://dx.doi.org/10.18632/aging.101641
dc.relationinfo:eu-repo/semantics/altIdentifier/url/https://www.aging-us.com/article/101641/text
dc.relationinfo:eu-repo/semantics/altIdentifier/url/https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6286822/
dc.rightshttps://creativecommons.org/licenses/by/2.5/ar/
dc.rightsinfo:eu-repo/semantics/openAccess
dc.subjectAMYLOID Β (AΒ)
dc.subjectAMYLOID Β PRECURSOR PROTEIN (APP)
dc.subjectGALLEIN
dc.subjectGO PROTEIN
dc.subjectGΒΓ COMPLEX
dc.titleAPP signaling in Alzheimer's disease
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:ar-repo/semantics/artículo
dc.typeinfo:eu-repo/semantics/publishedVersion


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