dc.contributorNORTHWESTERN UNIV
dc.contributorUniversidade Federal de São Paulo (UNIFESP)
dc.creatorCravo, S. L.
dc.creatorMorrison, S. F.
dc.date.accessioned2016-01-24T11:40:14Z
dc.date.accessioned2022-10-07T20:57:57Z
dc.date.available2016-01-24T11:40:14Z
dc.date.available2022-10-07T20:57:57Z
dc.date.created2016-01-24T11:40:14Z
dc.date.issued1993-09-03
dc.identifierBrain Research. Amsterdam: Elsevier B.V., v. 621, n. 1, p. 133-136, 1993.
dc.identifier0006-8993
dc.identifierhttp://repositorio.unifesp.br/handle/11600/25353
dc.identifier10.1016/0006-8993(93)90308-A
dc.identifierWOS:A1993LU74500018
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/4025438
dc.description.abstractThe caudal ventrolateral medulla (CVLM) contains neurons that are essential for the elaboration of the sympathoinhibitory effects of baroreceptor afferent stimulation. To determine if neurons in the CVLM also mediate a tonic inhibition of sympathetic nerve activity (SNA), we examined the effects of lesioning CVLM neurons in baroreceptor-denervated rats. the sustained increases in both arterial pressure (AP; 40 mmHg) and splanchnic SNA (200%) indicate that the discharge of neurons in the CVLM maintains a tonic sympathoinhibition that contributes significantly to the maintenance of normotensive levels of SNA and AP.
dc.languageeng
dc.publisherElsevier B.V.
dc.relationBrain Research
dc.rightshttp://www.elsevier.com/about/open-access/open-access-policies/article-posting-policy
dc.rightsAcesso restrito
dc.subjectVENTROLATERAL MEDULLA
dc.subjectSYMPATHETIC NERVE ACTIVITY
dc.subjectBLOOD PRESSURE REGULATION
dc.subjectBARORECEPTOR REFLEX
dc.titleTHE CAUDAL VENTROLATERAL MEDULLA IS A SOURCE of TONIC SYMPATHOINHIBITION
dc.typeArtigo


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