dc.creatorTardy, Marcienne Bloch
dc.date.accessioned2016-09-13T13:19:14Z
dc.date.accessioned2022-10-07T20:05:58Z
dc.date.available2016-09-13T13:19:14Z
dc.date.available2022-10-07T20:05:58Z
dc.date.created2016-09-13T13:19:14Z
dc.date.issued2003-01
dc.identifierTARDY, M. Brain aging: insights into neuron-glia interactions. R. Ci. méd. biol., Salvador, v. 2, n. 1, p. 114-122, jan./jun. 2003
dc.identifier2236-5222
dc.identifierhttp://repositorio.ufba.br/ri/handle/ri/20318
dc.identifierv.2, n.1
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/4016240
dc.description.abstractAging begins at maturity and is characterized by increasing deviations from an ideal functional state. One major reason for this is thought to be oxydatif stress. The brain is particularly susceptible to oxidative damage. Besides neurons, glial cells, a major family of neural cells, are directly involved in vital brain functions and particularly in the brain antioxidant defence. This review brings insights into the potentialities of these glial cells to protect neurons and into their contribution to the brain aging process.
dc.languagept_BR
dc.publisherInstituto de Ciências da Saúde/ Universidade Federal da Bahia
dc.rightsAcesso Aberto
dc.sourcehttp://www.portalseer.ufba.br/index.php/cmbio/issue/view/496/showToc
dc.subjectGlial function
dc.subjectOxidative stress
dc.subjectNeuroprotection
dc.subjectAging
dc.titleBrain aging: insights into neuron-glia interactions
dc.typeArtigo de Periódico


Este ítem pertenece a la siguiente institución