dc.creatorShantsila, Eduard
dc.creatorLip, Gregory YH
dc.date.accessioned2021-04-15T17:36:39Z
dc.date.accessioned2022-09-23T18:15:37Z
dc.date.available2021-04-15T17:36:39Z
dc.date.available2022-09-23T18:15:37Z
dc.date.created2021-04-15T17:36:39Z
dc.identifier9783319040936
dc.identifierhttps://directory.doabooks.org/handle/20.500.12854/35698
dc.identifierhttp://hdl.handle.net/20.500.12010/18737
dc.identifier10.1007/978-3-319-25460-9
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/3498085
dc.description.abstractThere are several disorders which carry an increased risk of thrombosis, clots that interfere with normal circulation, including: venous thromboembolism (VTE), comprising both deep vein thrombosis (DVT) and pulmonary embolism (PE), atrial fibrillation (AF), acute coronary syndromes (ACS), valve disease and endocarditis, and conditions associated with a raised risk of ischemic stroke. Due to this increased risk, a number of thromboprophylactic medications target the coagulation cascade. Effective anticoagulation can be achieved by inhibiting different coagulation factors in the coagulation cascade.
dc.languageeng
dc.publisherSpringer Nature
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAbierto (Texto Completo)
dc.rightshttps://creativecommons.org/licenses/by-nc/4.0/
dc.subjectHematology
dc.subjectSurgery
dc.subjectCardiology
dc.titleNon-Vitamin K Antagonist Oral Anticoagulants: A Concise Guide


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