| dc.creator | Steinman, Gary | |
| dc.date.accessioned | 2020-08-21T20:08:17Z | |
| dc.date.accessioned | 2022-09-23T18:14:47Z | |
| dc.date.available | 2020-08-21T20:08:17Z | |
| dc.date.available | 2022-09-23T18:14:47Z | |
| dc.date.created | 2020-08-21T20:08:17Z | |
| dc.identifier | 0306-9877 | |
| dc.identifier | https://doi.org/10.1016/j.mehy.2020.109797 | |
| dc.identifier | http://hdl.handle.net/20.500.12010/12108 | |
| dc.identifier | https://doi.org/10.1016/j.mehy.2020.109797 | |
| dc.identifier.uri | http://repositorioslatinoamericanos.uchile.cl/handle/2250/3497943 | |
| dc.description.abstract | The current pandemic of Covid-19 has created a paradigm for possibly gaining greater insight in two conditions:
1) Inflammatory maladies in pregnancy, and.
2) The biology of IGF-1 in autism.
Studies since the beginning of this century have supported the view that IGF-1 deficiency in the neonate defines the basis of autism. As a result, it appears that
interleukin-6 in corona virus-based infections causes reduced defenses because of suppressed IGF-1, especially in older patients. This may also portend an increase of
autism in the offspring of gravidas currently affected severely by Covid-19. | |
| dc.language | eng | |
| dc.publisher | Medical Hypotheses | |
| dc.rights | info:eu-repo/semantics/embargoedAccess | |
| dc.rights | Acceso restringido | |
| dc.source | reponame:Expeditio Repositorio Institucional UJTL | |
| dc.source | instname:Universidad de Bogotá Jorge Tadeo Lozano | |
| dc.subject | COVID-19 | |
| dc.subject | Autism | |
| dc.title | COVID-19 and autism | |
