The diverse roles of RIP kinases in host-pathogen interactions

dc.creatorVen Eng, Vik
dc.creatorWemyss, Madeleine A.
dc.creatorPearson, Jaclyn S.
dc.date.accessioned2020-09-04T19:39:02Z
dc.date.accessioned2022-09-23T18:04:38Z
dc.date.available2020-09-04T19:39:02Z
dc.date.available2022-09-23T18:04:38Z
dc.date.created2020-09-04T19:39:02Z
dc.identifier1084-9521
dc.identifierhttps://doi.org/10.1016/j.semcdb.2020.08.005
dc.identifierhttp://hdl.handle.net/20.500.12010/12730
dc.identifierhttps://doi.org/10.1016/j.semcdb.2020.08.005
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/3494600
dc.description.abstractReceptor Interacting Protein Kinases (RIPKs) are cellular signaling molecules that are critical for homeostatic signaling in both communicable and non-communicable disease processes. In particular, RIPK1, RIPK2, RIPK3 and RIPK7 have emerged as key mediators of intracellular signal transduction including inflammation, autophagy and programmed cell death, and are thus essential for the early control of many diverse pathogenic organisms. In this review, we discuss the role of each RIPK in host responses to bacterial and viral pathogens, with a focus on studies that have used pathogen infection models rather than artificial stimulation with purified
dc.languageeng
dc.publisherSeminars in Cell and Developmental Biology
dc.rightsinfo:eu-repo/semantics/embargoedAccess
dc.rightsAcceso restringido
dc.sourcereponame:Expeditio Repositorio Institucional UJTL
dc.sourceinstname:Universidad de Bogotá Jorge Tadeo Lozano
dc.subjectRIP kinase
dc.subjectInflammation
dc.subjectCell death
dc.subjectBacterial infection
dc.subjectViral infection
dc.subjectPathogen
dc.titleThe diverse roles of RIP kinases in host-pathogen interactions


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