Remodelação cardíaca induzida pelo tabagismo (tabaquismo)

dc.contributorSIIC
dc.contributorUniversidade Estadual Paulista (Unesp)
dc.date.accessioned2014-05-27T11:22:45Z
dc.date.available2014-05-27T11:22:45Z
dc.date.created2014-05-27T11:22:45Z
dc.date.issued2007-12-01
dc.identifierSalud(i)Ciencia, v. 15, n. 7, p. 1136-1138, 2007.
dc.identifier1667-8982
dc.identifierhttp://hdl.handle.net/11449/70202
dc.identifier2-s2.0-38349129926
dc.identifier5016839015394547
dc.description.abstractPrevious studies accomplished by our group suggest that tobacco smoke exposure results in cardiac remodeling, with progressive ventricular dysfunction. However, the mechanisms involved in this process are not known. Therefore, our laboratory has been trying to identify the potentials responsible mechanisms for cardiac remodeling induced by tobacco. The blood pressure increase, the renin-angiotensin system and the oxidative stress can modulate this process. On the other hand, the activation of MMP-2 or MMP-9, as well as lipid peroxidation, don't seem to participate of the morphologic and functional alterations induced by tobacco smoke exposure.
dc.languagepor
dc.relationSalud(i)Ciencia
dc.relation0,103
dc.rightsAcesso restrito
dc.sourceScopus
dc.subjectMetalloproteinases
dc.subjectOxidative stress
dc.subjectRenin-angiotensin system
dc.subjectVentricular function
dc.subjectVentricular remodeling
dc.subjectgelatinase A
dc.subjectgelatinase B
dc.subjectadrenergic system
dc.subjectblood pressure measurement
dc.subjectenzyme activation
dc.subjectheart ventricle function
dc.subjectheart ventricle remodeling
dc.subjecthuman
dc.subjectlipid peroxidation
dc.subjectmorphology
dc.subjectoxidative stress
dc.subjectrenin angiotensin aldosterone system
dc.subjecttobacco
dc.titleRemodelação cardíaca induzida pelo tabagismo (tabaquismo)
dc.typeArtículos de revistas


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