Effect of nicotinic stimulation of the amygdaloid complex on water and sodium chloride intake

dc.contributorUniversidade Estadual Paulista (Unesp)
dc.date.accessioned2014-05-27T11:17:58Z
dc.date.available2014-05-27T11:17:58Z
dc.date.created2014-05-27T11:17:58Z
dc.date.issued1994-12-01
dc.identifierRevista de Ciencias Biomedicas, v. 15, p. 41-48.
dc.identifier0101-322X
dc.identifierhttp://hdl.handle.net/11449/64540
dc.identifier2-s2.0-0028658470
dc.identifier1023597870118105
dc.identifier6551236936295697
dc.description.abstractWe studied the nicotine stimulation of the amygdaloid complex (AMG) on sodium and water intake in satiated and water-deprived rats. Nicotine produced no change in sodium or water intake in satiated animals when injected directly into the AMG. In water-deprived animals, nicotine injected into the AMG (basolateral nuclei) only blocked sodium chloride intake. We have previously demontrated that carbachol inhibits water and sodium intake in both satiated and water-deprived animals injected into the AMG. Injection of hexamethonium into the AMG totally blocked water intake in satiated and water-deprived animals. Hexamethonium injected into the AMG prior to nicotine produced no change in sodium intake. Thus, the present data suggest that sodium and water intake are mediated by a specific population of cholinoceptive neurons in the amygdaloid complex.
dc.languageeng
dc.relationRevista de Ciencias Biomedicas
dc.rightsAcesso restrito
dc.sourceScopus
dc.subjectamygdaloid complex
dc.subjecthexamethonium
dc.subjectnicotine
dc.subjectsodium appetite
dc.subjectwater intake
dc.subjectcarbachol
dc.subjectamygdaloid nucleus
dc.subjectanimal experiment
dc.subjectcholinergic system
dc.subjectcontrolled study
dc.subjectdrug effect
dc.subjectfluid intake
dc.subjectintracerebroventricular drug administration
dc.subjectmale
dc.subjectnonhuman
dc.subjectrat
dc.subjectsodium intake
dc.titleEffect of nicotinic stimulation of the amygdaloid complex on water and sodium chloride intake
dc.typeArtículos de revistas


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