dc.contributorResearch Incentive Fund of Hospital de Clínicas de Porto Alegre (FIPE-HCPA No. 12-0114)en-US
dc.contributorThe National Council for Scientific and Technological Development of Brazil (CNPq), Fund for Research Support of the State of Rio Grande do Sul (FAPERGS) and Programen-US
dc.creatorOchs de Muñoz, Gustavo Alfredo
dc.creatorKretzmann, Nelson Alexandre
dc.creatorLópez, Mónica Luján
dc.creatorKieling, Carlos Oscar
dc.creatorUribe-Cruz, Carolina
dc.creatorda Silva, Luciano Basso
dc.creatorOsvaldt, Alessandro Bersch
dc.creatorMatte, Ursula da Silveira
dc.date2017-01-17
dc.date.accessioned2018-11-07T18:54:38Z
dc.date.available2018-11-07T18:54:38Z
dc.identifierhttps://seer.ufrgs.br/hcpa/article/view/68336
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/2174827
dc.descriptionWe investigated the influence of bone marrow cells upon activation of ERK ½ in an animal model of 90% PH. Phosphorylated ERK 1/2 was evaluated by western blot. No differences were found between the groups. Thus, increased survival does not appear to be mediated by ERK 1/2 activation.Keywords: MAP Kinase Signaling System; stem cells; acute liver failureen-US
dc.formatapplication/pdf
dc.languageeng
dc.publisherHCPA/FAMED/UFRGSen-US
dc.relationhttps://seer.ufrgs.br/hcpa/article/view/68336/pdf
dc.relationhttps://seer.ufrgs.br/hcpa/article/downloadSuppFile/68336/38015
dc.rightsDireitos autorais 2017 Clinical and Biomedical Researchpt-BR
dc.rightshttp://creativecommons.org/licenses/by/4.0pt-BR
dc.sourceClinical & Biomedical Research; v. 36, n. 4 (2016)en-US
dc.sourceClinical and Biomedical Research; v. 36, n. 4 (2016)pt-BR
dc.source2357-9730
dc.source0101-5575
dc.subjecten-US
dc.subjectMAP Kinase Signaling System; stem cells; acute liver failureen-US
dc.subjecten-US
dc.titleERK 1/2 activation does not contribute to increased survival mediated by bone marrow cells after 90% partial hepatectomy in Wistar ratsen-US
dc.typeArtículos de revistas
dc.typeArtículos de revistas
dc.typePeer-reviewed Articleen-US


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