Suppression Of Dioxin Mediated Aryl Hydrocarbon Receptor Transformation By Ethanolic Extracts Of Propolis.

dc.creatorPark, Yong K
dc.creatorFukuda, Itsuko
dc.creatorAshida, Hitoshi
dc.creatorNishiumi, Shin
dc.creatorGuzman, Julio Paredes
dc.creatorSato, Helia H
dc.creatorPastore, Glaucia M
dc.date2004-Apr
dc.date2015-11-27T12:58:07Z
dc.date2015-11-27T12:58:07Z
dc.date.accessioned2018-03-29T00:58:58Z
dc.date.available2018-03-29T00:58:58Z
dc.identifierBioscience, Biotechnology, And Biochemistry. v. 68, n. 4, p. 935-8, 2004-Apr.
dc.identifier0916-8451
dc.identifier
dc.identifierhttp://www.ncbi.nlm.nih.gov/pubmed/15118327
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/195784
dc.identifier15118327
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1296017
dc.descriptionPresent study demonstrated that the ethanolic extracts of propolis containing higher concentrations of flavonoids suppressed 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced aryl hydrocarbon receptor transformation in a dose-dependent manner. The IC(50) values of propolis group 3 and group 12 were 1.2 and 3.6 microg/ml, respectively, indicating that propolis showed stronger antagonistic effects as compared with vegetable extracts.
dc.description68
dc.description935-8
dc.languageeng
dc.relationBioscience, Biotechnology, And Biochemistry
dc.relationBiosci. Biotechnol. Biochem.
dc.rightsfechado
dc.rights
dc.sourcePubMed
dc.subjectAnimals
dc.subjectDose-response Relationship, Drug
dc.subjectEthanol
dc.subjectFlavonoids
dc.subjectFruit
dc.subjectGene Expression Regulation
dc.subjectInhibitory Concentration 50
dc.subjectPlant Extracts
dc.subjectPropolis
dc.subjectRats
dc.subjectReceptors, Aryl Hydrocarbon
dc.subjectSuppression, Genetic
dc.subjectTetrachlorodibenzodioxin
dc.subjectVegetables
dc.titleSuppression Of Dioxin Mediated Aryl Hydrocarbon Receptor Transformation By Ethanolic Extracts Of Propolis.
dc.typeArtículos de revistas


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