Decerebrate Rigidity With Preserved Cognition And Gait: A Possible Role Of Anoxic-ischemic Brain Damage.

dc.creatorDamasceno, B P
dc.date1991-Jun
dc.date2015-11-27T12:18:44Z
dc.date2015-11-27T12:18:44Z
dc.date.accessioned2018-03-29T00:51:47Z
dc.date.available2018-03-29T00:51:47Z
dc.identifierThe International Journal Of Neuroscience. v. 58, n. 3-4, p. 283-7, 1991-Jun.
dc.identifier0020-7454
dc.identifier
dc.identifierhttp://www.ncbi.nlm.nih.gov/pubmed/1365051
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/193916
dc.identifier1365051
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1294149
dc.descriptionA case of stable decerebrate posture in the upper limbs following sudden loss of consciousness and prolonged coma is described. The patient recovered most of her cognitive functions and gait, without clinical, neurophysiological or neuroradiological evidence of brainstem lesion. MRI shows borderzone infarcts. It is suggested that anoxic-ischemic cortical damage, affecting specially corticoreticular neurons, could explain the development of decerebrate rigidity in patients without apparent brainstem lesion.
dc.description58
dc.description283-7
dc.languageeng
dc.relationThe International Journal Of Neuroscience
dc.relationInt. J. Neurosci.
dc.rightsfechado
dc.rights
dc.sourcePubMed
dc.subjectAdult
dc.subjectAnoxia
dc.subjectArm
dc.subjectBrain
dc.subjectBrain Ischemia
dc.subjectDecerebrate State
dc.subjectFemale
dc.subjectHumans
dc.subjectMagnetic Resonance Imaging
dc.subjectNeuropsychological Tests
dc.titleDecerebrate Rigidity With Preserved Cognition And Gait: A Possible Role Of Anoxic-ischemic Brain Damage.
dc.typeArtículos de revistas


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