dc.creatorCarvalho, AC
dc.creatorGuillaumon, AT
dc.creatorCintra, ED
dc.creatorde Figueiredo, LC
dc.creatorMoreira, MM
dc.creatorAraujo, S
dc.date2011
dc.dateJUL-SEP
dc.date2014-07-30T14:32:18Z
dc.date2015-11-26T16:47:25Z
dc.date2014-07-30T14:32:18Z
dc.date2015-11-26T16:47:25Z
dc.date.accessioned2018-03-28T23:33:33Z
dc.date.available2018-03-28T23:33:33Z
dc.identifierRevista Brasileira De Cirurgia Cardiovascular. Soc Brasil Cirurgia Cardiovasc, v. 26, n. 3, n. 404, n. 412, 2011.
dc.identifier0102-7638
dc.identifierWOS:000297233800015
dc.identifier10.5935/1678-9741.20110015
dc.identifierhttp://www.repositorio.unicamp.br/jspui/handle/REPOSIP/59916
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/59916
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1274854
dc.descriptionObjectives: To evaluate plasmatic arginine vasopressin (AVP) levels in patients undergoing scheduled conventional abdominal aortic aneurysm (AAA) repair. Methods: Plasmatic AVP concentrations were measured by radioimmunoassay in 22 non-consecutive adult patients undergoing infra-renal AAA repair. They were under combined general and epidural anesthesia at the following time frames: 1 - pre-operative (TO); 2 - 2h (Ti) and 6h (T2) after the surgical procedure; 3 - in the morning at the first (T3), second (T4) and third (TS) post-operative days. Some clinical and laboratory variables were also recorded. Results: The mean age of patients was 68 +/- 10 years; 17 were males. Plasmatic AVP (mean +/- SD; pg/mL) was within the normal range at T0 (1.4 +/- 0.7; baseline), increasing significantly at T1 (62.6 +/- 62.9; P<0.001) and at T2 (31.5 +/- 49.7; P<0.001), with a progressive fall, returning to basal levels at (2.1 +/- 3.8; P=NS). Positive and statistically significant correlations were found between AVP and glycemia, serum lactate and white blood cells counts, but not with systemic arterial pressure or plasma osmolarity during the postoperative period. Conclusions: Considering that no correlations were found between AVP levels and hemodynamic or plasmatic osmolarity variations in AAA repair, it seems that stress response is mainly secondary to noxious stimulation mediated by the autonomic nervous system that is not completely blocked by anesthetics.
dc.description26
dc.description3
dc.description404
dc.description412
dc.languageen
dc.publisherSoc Brasil Cirurgia Cardiovasc
dc.publisherSao Paulo Sp
dc.publisherBrasil
dc.relationRevista Brasileira De Cirurgia Cardiovascular
dc.relationRev. Bras. Cir. Cardiovasc.
dc.rightsaberto
dc.sourceWeb of Science
dc.subjectIntensive care units
dc.subjectCardiovascular abnormalities
dc.subjectReceptors
dc.subjectvasopressin
dc.subjectStress-response
dc.subjectControlled-trial
dc.subjectSurgical Stress
dc.subjectSurgery
dc.subjectHormone
dc.subjectSurveillance
dc.subjectAnesthesia
dc.subjectIsoflurane
dc.subjectOperations
dc.subjectCopeptin
dc.titlePlasmatic vasopressin in patients undergoing conventional infra-renal abdominal aorta aneurysm repair
dc.typeArtículos de revistas


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