dc.creatorMedeiros, CCM
dc.creatorde Lemos-Marini, SHV
dc.creatorBricola, M
dc.creatorCamargo, EE
dc.creatorSantos, AO
dc.creatorMagna, LA
dc.creatorGuerra, G
dc.creatorBaptista, MTM
dc.creatorMaciel-Guerra, AT
dc.date2009
dc.dateFEB
dc.date2014-11-14T05:57:05Z
dc.date2015-11-26T16:04:45Z
dc.date2014-11-14T05:57:05Z
dc.date2015-11-26T16:04:45Z
dc.date.accessioned2018-03-28T22:53:52Z
dc.date.available2018-03-28T22:53:52Z
dc.identifierJournal Of Pediatric Endocrinology & Metabolism. Freund Publishing House Ltd, v. 22, n. 2, n. 109, n. 118, 2009.
dc.identifier0334-018X
dc.identifierWOS:000264855400003
dc.identifierhttp://www.repositorio.unicamp.br/jspui/handle/REPOSIP/77100
dc.identifierhttp://www.repositorio.unicamp.br/handle/REPOSIP/77100
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/77100
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1265563
dc.descriptionCoordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
dc.descriptionAlthough autoimmune thyroid disease (AITD) is frequent in Turner's syndrome (TS), follow-up studies are scant, and there are none regarding subclinical thyroiditis. We investigated thyroid function and morphology in 17 patients with TS (mean age 14.6 years) with transient and asymptomatic variations of TSH and/or thyroid hormones. Our 2-year follow-up included measurements of TSH, free T(4), T(3) and TPO and Tg antibodies, ultrasound (US) (first and last evaluations) and scintigraphy (first evaluation). Thyroid volume was evaluated relative to the patients' stature. Fourteen had abnormal hormones, including four with hypothyroidism and one with hyperthyroidism, ten had positive antibodies, and all had abnormalities on US; uptake was normal in 14/16. Abnormal hormones were independent of antibodies, number of US findings, age, time of disease and volume. At the end of the follow-up, antibodies were associated with a high number of abnormal US features, particularly heterogeneous texture. Our results indicate that recurring thyroid hormone variations in TS are due to chronic AITD.
dc.descriptiono TEXTO COMPLETO DESTE ARTIGO, ESTARÁ DISPONÍVEL À PARTIR DE AGOSTO DE 2015.
dc.description22
dc.description2
dc.description109
dc.description118
dc.descriptionCoordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
dc.descriptionCoordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
dc.languageen
dc.publisherFreund Publishing House Ltd
dc.publisherTel Aviv
dc.publisherIsrael
dc.relationJournal Of Pediatric Endocrinology & Metabolism
dc.relationJ. Pediatr. Endocrinol. Metab.
dc.rightsembargo
dc.sourceWeb of Science
dc.subjectTurner's syndrome
dc.subjectautoimmune thyroid disease
dc.subjectultrasonography
dc.subjectscintigraphy
dc.subjectHypothyroidism
dc.subjectPrevalence
dc.subjectAntibodies
dc.subjectChildren
dc.subjectDysfunction
dc.subjectUltrasound
dc.subjectRecovery
dc.titleTurner's Syndrome and Subclinical Autoimmune Thyroid Disease: A Two-Year Follow-up Study
dc.typeArtículos de revistas


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