dc.creatorCroy B.A.
dc.creatorBurke S.D.
dc.creatorBarrette V.F.
dc.creatorZhang J.
dc.creatorHatta K.
dc.creatorSmith G.N.
dc.creatorBianco J.
dc.creatorYamada A.T.
dc.creatorAdams M.A.
dc.date2011
dc.date2015-06-30T20:24:50Z
dc.date2015-11-26T14:49:09Z
dc.date2015-06-30T20:24:50Z
dc.date2015-11-26T14:49:09Z
dc.date.accessioned2018-03-28T22:00:05Z
dc.date.available2018-03-28T22:00:05Z
dc.identifier
dc.identifierPregnancy Hypertension. , v. 1, n. 1, p. 87 - 94, 2011.
dc.identifier22107789
dc.identifier10.1016/j.preghy.2010.10.002
dc.identifierhttp://www.scopus.com/inward/record.url?eid=2-s2.0-78751526479&partnerID=40&md5=b193b62cf058e20b7ea80f5e8b3c5508
dc.identifierhttp://www.repositorio.unicamp.br/handle/REPOSIP/107842
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/107842
dc.identifier2-s2.0-78751526479
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1253814
dc.descriptionPre-eclampsia, an acute complication of human pregnancy, is associated with incomplete physiological modification of decidual spiral arteries. This is thought to promote oxidative stress from perfusion/reperfusion of the placenta and to restrict placental and fetal growth. Alymphoid (genotype Rag2 -/-/Il2rg-/-) mice, sufficient in dendritic and myeloid cell functions, lack spiral arterial modification with individual spiral arteries having ∼1.7× the vascular resistance and 0.66× the blood velocity of +/+ mice. Their placentae are not measurably hypoxic and neither placental growth nor fetal survival is impaired and gestational hypertension is not seen. Thus, lymphocytes rather than vascular adaptations appear to be the pivotal contributors to the clinical complications of pre-eclampsia. © 2010 International Society for the Study of Hypertension in Pregnancy. Published by Elsevier B.V. All rights reserved.
dc.description1
dc.description1
dc.description87
dc.description94
dc.descriptionRedman, C.W., Sargent, I.L., Latest advances in understanding preeclampsia (2005) Science, 308, pp. 1592-1594
dc.descriptionLyall, F., Belfort, M., (2007) Pre-eclampsia. Etiology and Clinical Practice, , Cambridge University Press Cambridge, UK
dc.descriptionPijnenborg, R., Dixon, G., Robertson, W.B., Brosens, I., Trophoblastic invasion of human decidua from 8 to 18 weeks of pregnancy (1980) Placenta, 1, pp. 3-19
dc.descriptionPijnenborg, R., Vercruysse, L., Hanssens, M., The uterine spiral arteries in human pregnancy: Facts and controversies (2006) Placenta, 27, pp. 939-958
dc.descriptionRedline, R.W., Patterson, P., Pre-eclampsia is associated with an excess of proliferative immature intermediate trophoblast (1995) Hum Pathol, 26, pp. 594-600
dc.descriptionKaufmann, P., Black, S., Huppertz, B., Endovascular trophoblast invasion: Implications for the pathogenesis of intrauterine growth retardation and preeclampsia (2003) Biol Reprod, 69, pp. 1-7
dc.descriptionLevine, R.J., Maynard, S.E., Qian, C., Circulating angiogenic factors and the risk of preeclampsia (2004) N Engl J Med, 350, pp. 672-683
dc.descriptionRomero, R., Nien, J.K., Espinoza, J., A longitudinal study of angiogenic (placental growth factor) and anti-angiogenic (soluble endoglin and soluble vascular endothelial growth factor receptor-1) factors in normal pregnancy and patients destined to develop preeclampsia and deliver a small for gestational age neonate (2008) J Matern Fetal Neonatal Med, 21, pp. 9-23
dc.descriptionMaynard, S., Epstein, F.H., Karumanchi, S.A., Preeclampsia and angiogenic imbalance (2008) Annu Rev Med, 59, pp. 61-78
dc.descriptionHerse, F., Verlohren, S., Wenzel, K., Prevalence of agonistic autoantibodies against the angiotensin II type 1 receptor and soluble fms-like tyrosine kinase 1 in a gestational age-matched case study (2009) Hypertension, 53, pp. 393-398
dc.descriptionHerse, F., Staff, A.C., Hering, L., AT1-receptor autoantibodies and uteroplacental RAS in pregnancy and pre-eclampsia (2008) J Mol Med, 86, pp. 697-703
dc.descriptionHiby, S.E., Walker, J.J., O'Shaughnessy, K.M., Combinations of maternal KIR and fetal HLA-C genes influence the risk of preeclampsia and reproductive success (2004) J Exp Med, 200, pp. 957-965
dc.descriptionFounds, S.A., Conley, Y.P., Lyons-Weiler, J.F., Altered global gene expression in first trimester placentas of women destined to develop preeclampsia (2009) Placenta, 30, pp. 15-24
dc.descriptionPeel, S., Granulated metrial gland cells (1989) Adv Anat Embryol Cell Biol, 115, pp. 1-112
dc.descriptionWang, B., Biron, C., She, J., A block in both early T lymphocyte and natural killer cell development in transgenic mice with high-copy numbers of the human CD3E gene (1994) Proc Natl Acad Sci USA, 91, pp. 9402-9406
dc.descriptionDisanto, J.P., Muller, W., Guy-Grand, D., Fischer, A., Rajewsky, K., Lymphoid development in mice with a targeted deletion of the interleukin 2 receptor gamma chain (1995) Proc Natl Acad Sci USA, 92, pp. 377-381
dc.descriptionGuimond, M.J., Wang, B., Fujita, J., Terhorst, C., Croy, B.A., Pregnancy-associated uterine granulated metrial gland cells in mutant and transgenic mice (1996) Am J Reprod Immunol, 35, pp. 501-509
dc.descriptionGreenwood, J.D., Minhas, K., Di Santo, J.P., Ultrastructural studies of implantation sites from mice deficient in uterine natural killer cells (2000) Placenta, 21, pp. 693-702
dc.descriptionGuimond, M.J., Wang, B., Croy, B.A., Engraftment of bone marrow from severe combined immunodeficient (SCID) mice reverses the reproductive deficits in natural killer cell-deficient tg epsilon 26 mice (1998) J Exp Med, 187, pp. 217-223
dc.descriptionAshkar, A.A., Di Santo, J.P., Croy, B.A., Interferon gamma contributes to initiation of uterine vascular modification, decidual integrity, and uterine natural killer cell maturation during normal murine pregnancy (2000) J Exp Med, 192, pp. 259-270
dc.descriptionMurphy, S.P., Tayade, C., Ashkar, A.A., Interferon gamma in successful pregnancies (2009) Biol Reprod, 80, pp. 848-859
dc.descriptionGermain, S.J., Sacks, G.P., Sooranna, S.R., Sargent, I.L., Redman, C.W., Systemic inflammatory priming in normal pregnancy and preeclampsia: The role of circulating syncytiotrophoblast microparticles (2007) J Immunol, 178, pp. 5949-5956
dc.descriptionRedman, C.W., Sargent, I.L., Microparticles and immunomodulation in pregnancy and pre-eclampsia (2007) J Reprod Immunol, 76, pp. 61-67
dc.descriptionGammill, H.S., Roberts, J.M., Emerging concepts in preeclampsia investigation (2007) Front Biosci, 12, pp. 2403-2411
dc.descriptionBainbridge, S.A., Roberts, J.M., Von Versen-Hoynck, F., Uric acid attenuates trophoblast invasion and integration into endothelial cell monolayers (2009) Am J Physiol Cell Physiol, 297, pp. 440-C450
dc.descriptionHershkovitz, R., De Swiet, M., Kingdom, J., Mid-trimester placentation assessment in high-risk pregnancies using maternal serum screening and uterine artery Doppler (2005) Hypertens Pregnancy, 24, pp. 273-280
dc.descriptionBurton, G.J., Jauniaux, E., Placental oxidative stress: From miscarriage to preeclampsia (2004) J Soc Gynecol Invest, 11, pp. 342-352
dc.descriptionThachil, J., Toh, C.H., Disseminated intravascular coagulation in obstetric disorders and its acute haematological management (2009) Blood Rev, 23, pp. 167-176
dc.descriptionRoberts, J.M., Gammill, H., Pre-eclampsia and cardiovascular disease in later life (2005) Lancet, 366, pp. 961-962
dc.descriptionSmith, G.N., Walker, M.C., Liu, A., A history of preeclampsia identifies women who have underlying cardiovascular risk factors (2009) Am J Obstet Gynecol, 200
dc.descriptionBurke, S.D., Barrette, V.F., Bianco, J., Spiral arterial remodeling is not essential for normal blood pressure regulation in pregnant mice (2010) Hypertension, 55
dc.descriptionMu, J., Slevin, J.C., Qu, D., McCormick, S., Adamson, S.L., In vivo quantification of embryonic and placental growth during gestation in mice using micro-ultrasound (2008) Reprod Biol Endocrinol, 6, p. 34
dc.descriptionZhang, J., Croy, B.A., Using ultrasonography to define fetal-maternal relationships: Moving from humans to mice (2009) Comp Med, 59, pp. 527-533
dc.descriptionPal, S., Peterson, E.M., De La Maza, L.M., A murine model for the study of Chlamydia trachomatis genital infections during pregnancy (1999) Infect Immun, 67, pp. 2607-2610
dc.descriptionBuzas, E.I., Hollo, K., Rubliczky, L., Effect of pregnancy on proteoglycan-induced progressive polyarthritis in BALB/c mice: Remission of disease activity (1993) Clin Exp Immunol, 94, pp. 252-260
dc.descriptionZhang, J., Dong, H., Wang, B., Zhu, S., Croy, B.A., Dynamic changes occur in patterns of endometrial EFNB2/EPHB4 expression during the period of spiral arterial modification in mice (2008) Biol Reprod, 79, pp. 450-458
dc.descriptionGuzik, T.J., Hoch, N.E., Brown, K.A., Role of the T cell in the genesis of angiotensin II induced hypertension and vascular dysfunction (2007) J Exp Med, 204, pp. 2449-2460
dc.descriptionHoch, N.E., Guzik, T.J., Chen, W., Regulation of T-cell function by endogenously produced angiotensin II (2009) Am J Physiol Regul Integr Comp Physiol, 296, pp. 208-R216
dc.descriptionJurewicz, M., McDermott, D.H., Sechler, J.M., Human T and natural killer cells possess a functional renin-angiotensin system: Further mechanisms of angiotensin II-induced inflammation (2007) J Am Soc Nephrol, 18, pp. 1093-1102
dc.descriptionMadhur, M.S., Lob, H.E., McCann, L.A., Interleukin 17 promotes angiotensin II-induced hypertension, vascular dysfunction (2009) Hypertension
dc.descriptionHatta, K., Carter, A.L., Chen, Z., Leno-Durán, E., Ruiz-Ruiz, C., Olivares, E.G., Tse, M.Y., Croy, B.A., Dynamic expression of the vasoactive proteins Agtr1, Agtr2 and Nppa by mouse uterine Natural Killer cells Reprod Sci, , [Epub Oct.19]
dc.descriptionGuimond, M., Wang, B., Croy, B.A., Immune competence involving the natural killer cell lineage promotes placental growth (1999) Placenta, 20, pp. 441-450
dc.descriptionPaffaro Jr., V.A., Bizinotto, M.C., Joazeiro, P.P., Yamada, A.T., Subset classification of mouse uterine natural killer cells by DBA lectin reactivity (2003) Placenta, 24, pp. 479-488
dc.languageen
dc.publisher
dc.relationPregnancy Hypertension
dc.rightsfechado
dc.sourceScopus
dc.titleIdentification Of The Primary Outcomes That Result From Deficient Spiral Arterial Modification In Pregnant Mice
dc.typeArtículos de revistas


Este ítem pertenece a la siguiente institución