dc.creatorVercesi A.E.
dc.creatorKowaltowski A.J.
dc.creatorOliveira H.C.F.
dc.creatorCastilho R.F.
dc.date2006
dc.date2015-06-30T18:15:02Z
dc.date2015-11-26T14:28:31Z
dc.date2015-06-30T18:15:02Z
dc.date2015-11-26T14:28:31Z
dc.date.accessioned2018-03-28T21:31:42Z
dc.date.available2018-03-28T21:31:42Z
dc.identifier
dc.identifierFrontiers In Bioscience. , v. 11, n. SUPPL. 2, p. 2554 - 2564, 2006.
dc.identifier10939946
dc.identifier10.2741/1990
dc.identifierhttp://www.scopus.com/inward/record.url?eid=2-s2.0-33744492597&partnerID=40&md5=2c391498fb8a221f01ccee3890f229dc
dc.identifierhttp://www.repositorio.unicamp.br/handle/REPOSIP/103679
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/103679
dc.identifier2-s2.0-33744492597
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1246611
dc.descriptionMitochondrial Ca2+ transport is important in the maintenance of intracellular ion homeostasis, and also a key factor in the pathogenesis of many diseases. We discuss here the main aspects of mitochondrial Ca2+ transport, and how this transport is linked to changes in energy metabolism and redox state. Mitochondrial permeability transition, a consequence of excessive mitochondrial Ca2+ accumulation associated with oxidative stress is also discussed. Finally, our current understanding of the involvement of these mitochondrial processes in cardiac ischemia-reperfusion, neurodegeneration and dyslipidemias is presented.
dc.description11
dc.descriptionSUPPL. 2
dc.description2554
dc.description2564
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dc.languageen
dc.publisher
dc.relationFrontiers in Bioscience
dc.rightsfechado
dc.sourceScopus
dc.titleMitochondrial Ca2+ Transport, Permeability Transition And Oxidative Stress In Cell Death: Implications In Cardiotoxicity, Neurodegeneration And Dyslipidemias
dc.typeArtículos de revistas


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