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Nestin+cells forming spheroids aggregates resembling tumorspheres in experimental ENU-induced gliomas
(Histology and Histopathology, 2016)
Association of Notch-1, osteopontin and stem-like cells in ENUglioma malignant process
(Impact Journals LLC, 2018)
Cloning of glucocorticoid-regulated genes in C6/ST1 rat glioma phenotypic reversion
(1996-01-01)
The C6 rat glioma cell line is responsive to glucocorticoid hormones. C6 variants that are hyper-responsive (ST1) and resistant (P7) to hormone treatment have been derived previously. Glucocorticoid treatment of ST1 cells ...
Cloning of glucocorticoid-regulated genes in C6/ST1 rat glioma phenotypic reversion
(1996-01-01)
The C6 rat glioma cell line is responsive to glucocorticoid hormones. C6 variants that are hyper-responsive (ST1) and resistant (P7) to hormone treatment have been derived previously. Glucocorticoid treatment of ST1 cells ...
Dynamic interaction between glioma cells and their microenvironment: targeting Rac1 as a therapeutic approach
(Smart Science and Technology, 2015-07)
Rac1 has been described as a major player in glioma invasion and progression. The development of novel anti Rac1 inhibitors, such as 1A-116, provides a targeted rational approach to counteract glioma progression. This ...
Loss of 5 '-Methylthioadenosine Phosphorylase (MTAP) is Frequent in High-Grade Gliomas; Nevertheless, it is Not Associated with Higher Tumor Aggressiveness
(Mdpi, 2020-02-01)
The 5'-methylthioadenosine phosphorylase (MTAP) gene is located in the chromosomal region 9p21. MTAP deletion is a frequent event in a wide variety of human cancers; however, its biological role in tumorigenesis remains ...
Establishment and partial characterization of a continuous human malignant glioma cell line: NG97
(Kluwer Academic/plenum PublNew YorkEUA, 2001)
p53 Mutant Human Glioma Cells Are Sensitive to UV-C-Induced Apoptosis Due to Impaired Cyclobutane Pyrimidine Dimer Removal
(AMER ASSOC CANCER RESEARCH, 2009)
The p53 protein is a key regulator of cell responses to DNA damage, and it has been shown that It sensitizes glioma cells to the alkylating agent temozolomide by up-regulating the extrinsic apoptotic pathway, whereas it ...