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Miro1 as a novel regulator of hypertrophy in neonatal rat cardiomyocytes
(Elsevier, 2020)
Cardiac hypertrophy is an adaptive response to manage an excessive cardiac workload and maintain normal cardiac function. However, sustained hypertrophy leads to cardiomyopathy, cardiac failure, and death. Adrenergic ...
Estimation of the fractional sarcoplasmic reticulum Ca2+ release in intact cardiomyocytes using integrated Ca2+ fluxes
(General Physiol And BiophysicsBratislavaEslováquia, 2012)
Increased spontaneous activity and reduced inotropic response to catecholamines in ventricular myocytes from footshock-stressed rats
(Taylor & Francis LtdAbingdonInglaterra, 2010)
Effect of geraniol on rat cardiomyocytes and its potential use as a cardioprotective natural compound
(Pergamon-Elsevier Science Ltd, 2017-01)
Aims Reactive oxygen species (ROS) are generated in the ischaemic myocardium especially during early reperfusion and affect myocardial function and viability. Monoterpenes have been proposed to play beneficial roles in ...
Mechanical stretch increases L-type calcium channel stability in cardiomyocytes through a polycystin-1/AKT-dependent mechanism
(Elsevier Science BV, 2018)
The L-type calcium channel (LTCC) is an important determinant of cardiac contractility. Therefore, changes in LTCC activity or protein levels could be expected to affect cardiac function. Several studies describing LTCC ...
Development of cardiomyocyte hypotrophy in rats under prolonged treatment with a low dose of a nitric oxide synthesis inhibitor
(Elsevier Science BvAmsterdamHolanda, 2000)
Effects of Growth Hormone on Cardiac Remodeling During Resistance Training in Rats
(Arquivos Brasileiros Cardiologia, 2016-01-01)
Background: Although the beneficial effects of resistance training (RT) on the cardiovascular system are well established, few studies have investigated the effects of the chronic growth hormone (GH) administration on ...
Polycystin-1 Is a Cardiomyocyte Mechanosensor That Governs L-Type Ca2+ Channel Protein Stability
(2015)
Background-L-type calcium channel activity is critical to afterload-induced hypertrophic growth of the heart. However, the mechanisms governing mechanical stress-induced activation of L-type calcium channel activity are ...