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Hypochlorous acid inhibition by acetoacetate: Implications on neutrophil functions
(2004-08-01)
Type-1 diabetic patients experience hyperketonemia caused by an increase in fatty acid metabolism. Thus, the aim of this study was to measure the effect of ketone bodies as suppressors of oxidizing species produced by ...
Hypochlorous acid inhibition by acetoacetate: Implications on neutrophil functions
(2004-08-01)
Type-1 diabetic patients experience hyperketonemia caused by an increase in fatty acid metabolism. Thus, the aim of this study was to measure the effect of ketone bodies as suppressors of oxidizing species produced by ...
Molecular analysis of chronic granulomatous disease caused by defects in gp91-phox
(Wiley-lissNew YorkEUA, 1999)
BAY 41-2272, a soluble guanylate cyclase agonist, activates human mononuclear phagocytes
(Wiley-BlackwellHoboken, 2012)
Chronic granulomatous disease in Latin American patients: Clinical spectrum and molecular genetics
(Wiley-lissHobokenEUA, 2006)
Neutrophil dysfunction induced by hyperglycemia: modulation of myeloperoxidase activity
(WILEY-BLACKWELLHOBOKEN, 2013-08-02)
Our data suggest that impaired activity of myeloperoxidase (MPO) may play an important role in the dysfunction of neutrophils from hyperglycemic rats. Neutrophil biochemical pathways include the NADPH oxidase system and ...
Oxidation of lysergic acid diethylamide (LSD) by peroxidases: a new metabolic pathway
(SPRINGERNEW YORK, 2012)
Lysergic acid diethylamide (LSD) is a potent hallucinogen that is primarily metabolized to 2-oxo-3-hydroxy-LSD (O-H-LSD) and N-desmethyl-LSD (nor-LSD) by cytochrome P450 complex liver enzymes. Due to its extensive metabolism, ...