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The Cross-talk Between Angiotensin And Insulin Differentially Affects Phosphatidylinositol 3-kinase- And Mitogen-activated Protein Kinase-mediated Signaling In Rat Heart: Implications For Insulin Resistance.

Carvalheira, José B C; Calegari, Vivian C; Zecchin, Henrique G; Nadruz, Wilson; Guimarães, Regina B; Ribeiro, Eliane B; Franchini, Kleber G; Velloso, Lício A; Saad, Mario J A (2003)

Early Activation Of The Multicomponent Signaling Complex Associated With Focal Adhesion Kinase Induced By Pressure Overload In The Rat Heart.

Franchini, K G; Torsoni, A S; Soares, P H; Saad, M J (2000)

Role of c-jun N-terminal kinases (JNKs) in epilepsy and metabolic cognitive impairment

Busquets O.; Ettcheto M.; Cano A.; Manzine P.R.; Sánchez-Lopez E.; Espinosa-Jiménez T.; Verdaguer E.; Castro-Torres R.D.; Beas-Zarate C.; Sureda F.X.; Olloquequi J.; Auladell C.; Folch J.; Camins A. (MDPI AG, 2020)

Cardiovascular remodeling induced by passive smoking

Universidade Estadual Paulista (Unesp) (2009-12-01)

Coronary heart disease (CHD) is the most common cause of death in many developed countries. The major risk factors for CHD are smoking, high blood pressure, diabetes, high cholesterol levels, and lack of physical activity. ...

Propolis and its constituent caffeic acid suppress LPS-stimulated pro-inflammatory response by blocking NF-κB and MAPK activation in macrophages

Universidade Estadual Paulista (Unesp); Azinhaga de Santa Comba (2013-08-26)

Ethnopharmacological relevance Propolis is a bee product with numerous biological and pharmacological properties, such as immunomodulatory and anti-inflammatory activities. It has been used in folk medicine as a healthy ...

Propolis and its constituent caffeic acid suppress LPS-stimulated pro-inflammatory response by blocking NF-κB and MAPK activation in macrophages

Universidade Estadual Paulista (Unesp); Azinhaga de Santa Comba (2013-08-26)

Role of JNK isoforms in the kainic acid experimental model of epilepsy and neurodegeneration

Auladell C.; De Lemos L.; Verdaguer E.; Ettcheto M.; Busquets O.; Lazarowski A.; Beas-Zarate C.; Olloquequi J.; Folch J.; Camins A. (Frontiers in Bioscience, 2017)

Hyperglycaemia inhibits thymidine incorporation and cell growth via protein kinase c, mitogen-activated protein kinases and nitric oxide in human umbilical vein endothelium

Autor desconocido (CAMBRIDGE UNIVERSITY PRESS, 2003)

Regulation of adenosine transport by d-glucose in human fetal endothelial cells: involvement of nitric oxide, protein kinase c and mitogen-activated protein kinase

Autor desconocido (CAMBRIDGE UNIVERSITY PRESS, 2000)

Molecular Mechanism Of Violacein-mediated Human Leukemia Cell Death.

Ferreira, Carmen Veríssima; Bos, Carina L; Versteeg, Henri H; Justo, Giselle Z; Durán, Nelson; Peppelenbosch, Maikel P (2004)

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The Cross-talk Between Angiotensin And Insulin Differentially Affects Phosphatidylinositol 3-kinase- And Mitogen-activated Protein Kinase-mediated Signaling In Rat Heart: Implications For Insulin Resistance.

Early Activation Of The Multicomponent Signaling Complex Associated With Focal Adhesion Kinase Induced By Pressure Overload In The Rat Heart.

Role of c-jun N-terminal kinases (JNKs) in epilepsy and metabolic cognitive impairment

Cardiovascular remodeling induced by passive smoking

Propolis and its constituent caffeic acid suppress LPS-stimulated pro-inflammatory response by blocking NF-κB and MAPK activation in macrophages

Propolis and its constituent caffeic acid suppress LPS-stimulated pro-inflammatory response by blocking NF-κB and MAPK activation in macrophages

Role of JNK isoforms in the kainic acid experimental model of epilepsy and neurodegeneration

Hyperglycaemia inhibits thymidine incorporation and cell growth via protein kinase c, mitogen-activated protein kinases and nitric oxide in human umbilical vein endothelium

Regulation of adenosine transport by d-glucose in human fetal endothelial cells: involvement of nitric oxide, protein kinase c and mitogen-activated protein kinase

Molecular Mechanism Of Violacein-mediated Human Leukemia Cell Death.