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Toll-like receptor 4 activation prevents rat cardiac fibroblast death induced by simulated ischemia/reperfusion
(Frontiers Media, 2021)
Death of cardiac fibroblasts (CFs) by ischemia/reperfusion (I/R) has major implications
for cardiac wound healing. In in vivo models of myocardial infarction, toll-like receptor 4
(TLR4) activation has been reported as ...
Effect of retinal ischemia on the non-image forming visual system
(Taylor & Francis, 2014-09)
Retinal ischemic injury is an important cause of visual impairment. The loss of retinal ganglion cells (RGCs) is a key sign of retinal ischemic damage. A subset of RGCs expressing the photopigment melanopsin (mRGCs) regulates ...
Drp1 loss-of-function reduces cardiomyocyte oxygen dependence protecting the heart from ischemia-reperfusion injury
(Lippincott Williams and Wilkins, 2014)
Mitochondria are key organelles for ATP production in cardiomyocytes, which is regulated by processes of fission and fusion. We hypothesized that the mitochondria fusion protein dynamin-related protein 1 (Drp1) inhibition, ...
L-NIL prevents the ischemia and reperfusion injury involving TRL4, GST, clusterin and NFAT5 in mice
(American journal of physiology. Renal physiology, 2020)
Inhibition of the proteasome preserves Mitofusin-2 and mitochondrial integrity, protecting cardiomyocytes during ischemia-reperfusion injury
(Elsevier, 2020)
Cardiomyocyte loss is the main cause of myocardial dysfunction following an ischemia-reperfusion (IR) injury. Mitochondrial dysfunction and altered mitochondrial network dynamics play central roles in cardiomyocyte death. ...
L-NIL prevents the ischemia and reperfusion injury involving TLR-4, GST, clusterin, and NFAT-5 in mice
(American Physiological Society, 2019)
Copyright © 2019 the American Physiological Society.—On renal ischemia-reperfusion (I/R) injury, recruitment of neutrophils during the inflammatory process promotes local generation of oxygen and nitrogen reactive species, ...
L-NIL prevents the ischemia and reperfusion injury involving TLR-4, GST, clusterin, and NFAT-5 in mice
(Facultad de Medicina y Ciencia, 2019)
On renal ischemia-reperfusion (I/R) injury, recruitment of neutrophils during the inflammatory process promotes local generation of oxygen and nitrogen reactive species, which, in turn, are likely to exacerbate tissue ...
L-NIL prevents the ischemia and reperfusion injury involving TRL4, GST, clusterin and NFAT5 in mice
(American journal of physiology. Renal physiology, 2020)
TGF-ß1 previene el estrés de retículo endoplásmico y la muerte celular inducida por isquemia/reperfusión simulada en fibroblastos cardiacos de rata
(Universidad de ChileCyberDocs, 2011)
En el corazón, los fibroblastos son las células más abundantes y tienen características estructurales y funcionales muy determinadas: a) actúan como sensores de los cambios del entorno reaccionando frente a estímulos ...